(Q40443282)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

title

Ca2+-induced permeability transition in human lymphoblastoid cell mitochondria from normal and Huntington's disease individuals (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

0 references

J. Timothy Greenamyre

series ordinal

3

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

author name string

Alexander V Panov

series ordinal

1

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

Serena Lund

series ordinal

2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

publication date

1 January 2005

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

Molecular and Cellular Biochemistry

9 January 2020

published in

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

volume

269

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

issue

1-2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

page(s)

143-152

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15786727%20AND%20SRC:MED&resulttype=core&format=json

https://scigraph.springernature.com/pub.10.1007/s11010-005-3454-9

9 January 2020

exact match

0 references

cites work

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Early mitochondrial calcium defects in Huntington's disease are a direct effect of polyglutamines.

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Decreased expression of striatal signaling genes in a mouse model of Huntington's disease

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Mitochondrial depolarization is not required for neuronal apoptosis

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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Calcium-induced cytochrome c release from CNS mitochondria is associated with the permeability transition and rupture of the outer membrane

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Adenine nucleotide translocase as a site of regulation by ADP of the rat liver mitochondria permeability to H+ and K+ ions

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Increased apoptosis of Huntington disease lymphoblasts associated with repeat length-dependent mitochondrial depolarization

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Cells exposed to a huntingtin fragment containing an expanded polyglutamine tract show no sign of ion channel formation: results arguing against the ion channel hypothesis

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Widespread expression of the human and rat Huntington's disease gene in brain and nonneural tissues

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Release of mitochondrial glutathione and calcium by a cyclosporin A-sensitive mechanism occurs without large amplitude swelling

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Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions.

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12 December 2020

Mitochondrial involvement in Parkinson's disease, Huntington's disease, hereditary spastic paraplegia and Friedreich's ataxia

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Mitochondrial dysfunction and reactive oxygen species in excitotoxicity and apoptosis: implications for the pathogenesis of neurodegenerative diseases

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Apoptosis in neurodegenerative diseases: the role of mitochondria

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12 December 2020

Expression of polyglutamine-expanded Huntingtin activates the SEK1-JNK pathway and induces apoptosis in a hippocampal neuronal cell line

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The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

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Powerful cyclosporin inhibition of calcium-induced permeability transition in brain mitochondria

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12 December 2020

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12 December 2020

Quantitative evaluation of the effects of mitochondrial permeability transition pore modifiers on accumulation of calcium phosphate: comparison of rat liver and brain mitochondria

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12 December 2020

Ca(2+)-dependent permeability transition and complex I activity in lymphoblast mitochondria from normal individuals and patients with Huntington's or Alzheimer's disease

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12 December 2020

Mg2+ control of respiration in isolated rat liver mitochondria.

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12 December 2020

Mitochondrial transport of cations: channels, exchangers, and permeability transition.

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12 December 2020

Mitochondria in organismal aging and degeneration

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12 December 2020

Inhibiting caspase cleavage of huntingtin reduces toxicity and aggregate formation in neuronal and nonneuronal cells

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12 December 2020

Wild-type huntingtin protects from apoptosis upstream of caspase-3.

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12 December 2020

Signal presequences increase mitochondrial permeability and open the multiple conductance channel

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12 December 2020

Mitochondria and neuronal survival

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12 December 2020

Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

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12 December 2020

The mitochondrial permeability transition pore and its role in cell death

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12 December 2020

In vitro effects of polyglutamine tracts on Ca2+-dependent depolarization of rat and human mitochondria: relevance to Huntington's disease.

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12 December 2020

Recent advances on the pathogenesis of Huntington's disease

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12 December 2020

Poly-L-glutamine forms cation channels: relevance to the pathogenesis of the polyglutamine diseases

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12 December 2020

Assessment of mitochondrial oxidative phosphorylation in patient muscle biopsies, lymphoblasts, and transmitochondrial cell lines

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12 December 2020

The relationship between free and total calcium concentrations in the matrix of liver and brain mitochondria.

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12 December 2020

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Prooxidants open both the mitochondrial permeability transition pore and a low-conductance channel in the inner mitochondrial membrane

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12 December 2020

The striatum is the most vulnerable region in the brain to mitochondrial energy compromise: a hypothesis to explain its specific vulnerability

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12 December 2020