(Q40629432)

English

Regulation of necrosis of H9c2 myogenic cells upon transient energy deprivation. Rapid deenergization of mitochondria precedes necrosis and is controlled by reactive oxygen species, stress kinase JNK, HSP72 and ARC.

scientific article published on 30 September 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Regulation of necrosis of H9c2 myogenic cells upon transient energy deprivation. Rapid deenergization of mitochondria precedes necrosis and is controlled by reactive oxygen species, stress kinase JNK, HSP72 and ARC. (English)

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

reactive oxygen species

1 reference

based on heuristic

inferred from title

object named as

Julia A Yaglom

1

0 references

Michael Y Sherman

object named as

Michael Y Sherman

4

0 references

Vladimir L. Gabai

object named as

Vladimir L Gabai

3

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author name string

Daryoush Ekhterae

2

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

language of work or name

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publication date

30 September 2003

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Journal of Biological Chemistry

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

278

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

50

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

50483-50496

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

Biochemical pathways of caspase activation during apoptosis

1 reference

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21 January 2018

More than one way to die: apoptosis, necrosis and reactive oxygen damage

1 reference

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21 January 2018

Necrosis: a specific form of programmed cell death?

1 reference

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Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion

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Prevention of kidney ischemia/reperfusion-induced functional injury and JNK, p38, and MAPK kinase activation by remote ischemic pretreatment

1 reference

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21 January 2018

Suppression of stress kinase JNK is involved in HSP72-mediated protection of myogenic cells from transient energy deprivation. HSP72 alleviates the stewss-induced inhibition of JNK dephosphorylation

1 reference

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21 January 2018

Serine/threonine protein kinases and apoptosis.

1 reference

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21 January 2018

Kinases: positive and negative regulators of apoptosis.

1 reference

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21 January 2018

Signal transduction by the JNK group of MAP kinases

1 reference

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21 January 2018

Hsp72 and stress kinase c-jun N-terminal kinase regulate the bid-dependent pathway in tumor necrosis factor-induced apoptosis

1 reference

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21 January 2018

JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis

1 reference

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21 January 2018

Mitochondrial control of cell death

1 reference

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Mitochondrial dysfunction in the pathogenesis of necrotic and apoptotic cell death

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Cytochrome c-dependent activation of caspase-3 by tumor necrosis factor requires induction of the mitochondrial permeability transition

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21 January 2018

The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release

1 reference

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21 January 2018

On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

1 reference

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21 January 2018

Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts.

1 reference

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21 January 2018

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

1 reference

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21 January 2018

ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells

1 reference

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21 January 2018

TAT protein transduction into isolated perfused hearts: TAT-apoptosis repressor with caspase recruitment domain is cardioprotective

1 reference

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21 January 2018

Apoptosis repressor with caspase domain inhibits cardiomyocyte apoptosis by reducing K+ currents

1 reference

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21 January 2018

The apoptotic regulatory protein ARC (apoptosis repressor with caspase recruitment domain) prevents oxidant stress-mediated cell death by preserving mitochondrial function

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M306903200

21 January 2018

Adenovirus-mediated gene transfer of a heat shock protein 70 (hsp 70i) protects against simulated ischemia

1 reference

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21 January 2018

Gene transfer of heat-shock protein 70 reduces infarct size in vivo after ischemia/reperfusion in the rabbit heart

1 reference

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21 January 2018

Transgenic mice expressing the human heat shock protein 70 have improved post-ischemic myocardial recovery

1 reference

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21 January 2018

Overexpression of heat shock protein 72 in transgenic mice decreases infarct size in vivo

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Cardioprotective effects of 70-kDa heat shock protein in transgenic mice

1 reference

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21 January 2018

Oxidative stress induces DNA fragmentation and caspase activation via the c-Jun NH2-terminal kinase pathway in H9c2 cardiac muscle cells.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M306903200

21 January 2018

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

1 reference

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21 January 2018

Mitochondrial reactive oxygen species in cell death signaling

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Heat shock proteins delivered with a virus vector can protect cardiac cells against apoptosis as well as against thermal or hypoxic stress

1 reference

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Protein-damaging stresses activate c-Jun N-terminal kinase via inhibition of its dephosphorylation: a novel pathway controlled by HSP72.

1 reference

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21 January 2018

The chaperone function of hsp70 is required for protection against stress-induced apoptosis

1 reference

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The calmodulin antagonist trifluoperazine in transient focal brain ischemia in rats. Anti-ischemic effect and therapeutic window

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21 January 2018

Inhibition of the type 1 inositol 1,4,5-trisphosphate-sensitive Ca2+ channel by calmodulin antagonists

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21 January 2018

Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide

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Mitochondria, oxygen free radicals, and apoptosis

1 reference

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Mitochondrial complex I inhibitor rotenone induces apoptosis through enhancing mitochondrial reactive oxygen species production

1 reference

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Reactive species mechanisms of cellular hypoxia-reoxygenation injury

1 reference

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21 January 2018

Identifiers

10.1074/JBC.M306903200

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

22 September 2017

ResearchGate publication ID

0 references

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