(Q40734726)

23 September 2017

Contribution of death receptor and mitochondrial pathways to Fas-mediated apoptosis in the prostatic carcinoma cell line PC3. (English)

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23 September 2017

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Natalya V Guseva

1

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23 September 2017

Agshin F Taghiyev

series ordinal

2

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23 September 2017

Oskar W Rokhlin

series ordinal

3

1 reference

23 September 2017

Michael B Cohen

series ordinal

4

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23 September 2017

1 June 2002

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23 September 2017

1 reference

23 September 2017

51

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23 September 2017

4

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23 September 2017

231-240

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Induction of apoptosis by cancer chemotherapy.

23 September 2017

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Signal transduction mediated by Bid, a pro-death Bcl-2 family proteins, connects the death receptor and mitochondria apoptosis pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Death receptors: signaling and modulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumor necrosis factor receptor-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Membrane oligomerization and cleavage activates the caspase-8 (FLICE/MACHalpha1) death signal

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Role of cytochrome c and dATP/ATP hydrolysis in Apaf-1-mediated caspase-9 activation and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Bid, a critical mediator for apoptosis induced by the activation of Fas/TNF-R1 death receptors in hepatocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Two CD95 (APO-1/Fas) signaling pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Differential modulation of apoptosis sensitivity in CD95 type I and type II cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

The role of c-FLIP in modulation of CD95-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Bcl-2 oncoprotein protects the human prostatic carcinoma cell line PC3 from TRAIL-mediated apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Pro-caspase-3 is a major physiologic target of caspase-8.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Cleavage of poly(ADP-ribose) polymerase by a proteinase with properties like ICE

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

The levels of MDM2 protein are decreased by a proteasome-mediated proteolysis prior to caspase-3-dependent pRb and PARP cleavages

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Apoptosis induction by caspase-8 is amplified through the mitochondrial release of cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Mechanisms of CD95 (APO-1/Fas)-mediated apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

Cytochrome c promotes caspase-9 activation by inducing nucleotide binding to Apaf-1

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FPROS.10095

21 January 2018

Identifiers

DOI

10.1002/PROS.10095

1 reference

23 September 2017

1 reference