(Q40793451)

23 September 2017

A role for mitochondrial Bak in apoptotic response to anticancer drugs (English)

1 reference

23 September 2017

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Wang GQ

1

1 reference

23 September 2017

Gastman BR

2

1 reference

23 September 2017

Wieckowski E

3

1 reference

23 September 2017

Goldstein LA

4

1 reference

23 September 2017

Gambotto A

5

1 reference

23 September 2017

Kim TH

6

1 reference

23 September 2017

Fang B

7

1 reference

23 September 2017

Rabinovitz A

8

1 reference

23 September 2017

Yin XM

9

1 reference

23 September 2017

Rabinowich H

10

1 reference

23 September 2017

language of work or name

English

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publication date

10 July 2001

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Journal of Biological Chemistry

23 September 2017

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23 September 2017

276

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23 September 2017

36

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23 September 2017

34307-34317

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Apoptotic pathways: the roads to ruin

23 September 2017

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Molecular characterization of mitochondrial apoptosis-inducing factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Mitochondrial control of cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

BCL-2 family members and the mitochondria in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bid, a critical mediator for apoptosis induced by the activation of Fas/TNF-R1 death receptors in hepatocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bax directly induces release of cytochrome c from isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Role of BAX in the apoptotic response to anticancer agents

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Lysosomal protease pathways to apoptosis. Cleavage of bid, not pro-caspases, is the most likely route

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Granzyme B-mediated cytochrome c release is regulated by the Bcl-2 family members bid and Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Initiation of apoptosis by granzyme B requires direct cleavage of bid, but not direct granzyme B-mediated caspase activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

BAX is required for neuronal death after trophic factor deprivation and during development

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bax-deficient mice with lymphoid hyperplasia and male germ cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

BAX frameshift mutations in cell lines derived from human haemopoietic malignancies are associated with resistance to apoptosis and microsatellite instability

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Induction of apoptosis by the Bcl-2 homologue Bak

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Cloning of a bcl-2 homologue by interaction with adenovirus E1B 19K

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Modulation of apoptosis by the widely distributed Bcl-2 homologue Bak

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

BID: a novel BH3 domain-only death agonist

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Evaluation of GAL4/TATA in vivo. Induction of transgene expression by adenovirally mediated gene codelivery

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Analysis of the membrane potential of rat- and mouse-liver mitochondria by flow cytometry and possible applications.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Regulated targeting of BAX to mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

On the voltage dependence of the mitochondrial permeability transition pore. A critical appraisal.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Mitochondria and cell death. Mechanistic aspects and methodological issues

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Withdrawal of IL-7 induces Bax translocation from cytosol to mitochondria through a rise in intracellular pH.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

bax-deficiency promotes drug resistance and oncogenic transformation by attenuating p53-dependent apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Mutant p53: epigenetic mutator of the T-cell receptor via induction of methylation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Aberrant expression and function of p53 in T-cells immortalized by HTLV-I Tax1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

Role of Bak in UV-induced apoptosis in skin cancer and abrogation by HPV E6 proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M103526200

21 January 2018

Identifiers

DOI

10.1074/JBC.M103526200

1 reference

23 September 2017

1 reference