(Q40952393)

25 September 2017

P53-dependent effects of RAS oncogene on chromosome stability and cell cycle checkpoints (English)

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25 September 2017

Agapova LS

1

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25 September 2017

Ivanov AV

2

1 reference

25 September 2017

Sablina AA

3

1 reference

25 September 2017

Kopnin PB

4

1 reference

25 September 2017

Sokova OI

5

1 reference

25 September 2017

Chumakov PM

6

1 reference

25 September 2017

Kopnin BP

7

1 reference

25 September 2017

language of work or name

English

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publication date

1 May 1999

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25 September 2017

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25 September 2017

18

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25 September 2017

20

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25 September 2017

3135-3142

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1202386

25 September 2017

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cites work

Chromosome changes caused by alterations of p53 expression

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

p53 controls both the G2/M and the G1 cell cycle checkpoints and mediates reversible growth arrest in human fibroblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Motility of intracellular particles in rat fibroblasts is greatly enhanced by phorbol ester and by over-expression of normal p21N-ras

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

ras oncogenes: their role in neoplasia

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

A p53-dependent mouse spindle checkpoint

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

The human Ha-ras oncogene induces genomic instability in murine fibroblasts within one cell cycle

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

p53-dependent inhibition of cyclin-dependent kinase activities in human fibroblasts during radiation-induced G1 arrest.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

WAF1, a potential mediator of p53 tumor suppression

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Participation of p53 cellular tumour antigen in transformation of normal embryonic cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

In vitro establishment is not a sufficient prerequisite for transformation by activated ras oncogenes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Abnormal centrosome amplification in the absence of p53.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Cell cycle control and cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Mutant p53 tumor suppressor alleles release ras-induced cell cycle growth arrest

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Rescue of cells from ras oncogene-induced growth arrest by a second, complementing, oncogene

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Cellular immortalization by a cDNA clone encoding the transformation-associated phosphoprotein p53

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Induction of p27Kip1 degradation and anchorage independence by Ras through the MAP kinase signaling pathway.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Cancer-susceptibility genes. Gatekeepers and caretakers

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

The spectrum of mutations at the p53 locus. Evidence for tissue-specific mutagenesis, selection of mutant alleles, and a "gain of function" phenotype

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Ras transformation results in an elevated level of cyclin D1 and acceleration of G1 progression in NIH 3T3 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Use of genetic suppressor elements to dissect distinct biological effects of separate p53 domains

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Cooperation between gene encoding p53 tumour antigen and ras in cellular transformation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Inhibitors of mammalian G1 cyclin-dependent kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Biological effects of a murine retrovirus carrying an activated N-ras gene of human origin

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Cell-specific effects of RAS oncogene and protein kinase C agonist TPA on P-glycoprotein function.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

The human papillomavirus-16 E6 oncoprotein decreases the vigilance of mitotic checkpoints.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

p21 is a universal inhibitor of cyclin kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

p21waf1 can block cells at two points in the cell cycle, but does not interfere with processive DNA-replication or stress-activated kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

Uncoupling of S phase and mitosis induced by anticancer agents in cells lacking p21

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

K-ras modulates the cell cycle via both positive and negative regulatory pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

inferred from DOI database lookup

7 January 2021

based on heuristic

Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

inferred from DOI database lookup

7 January 2021

based on heuristic

Transformation and stimulation of DNA synthesis in NIH-3T3 cells are a titratable function of normal p21N-ras expression.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202386

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.ONC.1202386

1 reference

Dimensions Publication ID

25 September 2017

0 references

1 reference