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English
In a flurry of PINK, mitochondrial bioenergetics takes a leading role in Parkinson's disease.
scientific article published on May 2009
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instance of
scholarly article
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
title
In a flurry of PINK, mitochondrial bioenergetics takes a leading role in Parkinson's disease
(English)
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
main subject
Parkinson's disease
1 reference
based on heuristic
inferred from title
author
Anne N. Murphy
series ordinal
1
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
author name string
Anne N Murphy
series ordinal
1
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
retrieved
18 October 2017
publication date
1 May 2009
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
number of pages
4
1 reference
based on heuristic
inferred from page(s)
published in
EMBO Molecular Medicine
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
volume
1
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
issue
2
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
page(s)
81-84
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
cites work
Parkinson's disease mutations in PINK1 result in decreased Complex I activity and deficient synaptic function
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
PINK1-associated Parkinson's disease is caused by neuronal vulnerability to calcium-induced cell death
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Silencing of PINK1 expression affects mitochondrial DNA and oxidative phosphorylation in dopaminergic cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
PINK1 defect causes mitochondrial dysfunction, proteasomal deficit and alpha-synuclein aggregation in cell culture models of Parkinson's disease
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Parkin, PINK1, and DJ-1 form a ubiquitin E3 ligase complex promoting unfolded protein degradation
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Pink1 forms a multiprotein complex with Miro and Milton, linking Pink1 function to mitochondrial trafficking
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Molecular basis of Parkinson's disease
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Loss-of-function analysis suggests that Omi/HtrA2 is not an essential component of the PINK1/PARKIN pathway in vivo
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Bidirectional Ca2+-dependent control of mitochondrial dynamics by the Miro GTPase
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
PINK1 controls mitochondrial localization of Parkin through direct phosphorylation
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
The Parkinson's disease genes pink1 and parkin promote mitochondrial fission and/or inhibit fusion in Drosophila
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
PINK1 is necessary for long term survival and mitochondrial function in human dopaminergic neurons
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Mitochondrial fusion, fission and autophagy as a quality control axis: the bioenergetic view
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Pink1 regulates mitochondrial dynamics through interaction with the fission/fusion machinery
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
The PINK1/Parkin pathway regulates mitochondrial morphology
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Loss-of-function of human PINK1 results in mitochondrial pathology and can be rescued by parkin
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
The mitochondrial protease HtrA2 is regulated by Parkinson's disease-associated kinase PINK1.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Mitochondrial dysfunction, peroxidation damage and changes in glutathione metabolism in PARK6.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Hereditary early-onset Parkinson's disease caused by mutations in PINK1
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Mechanistic approaches to Parkinson's disease pathogenesis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
26 October 2017
Mitochondrial respiratory dysfunction in familiar parkinsonism associated with PINK1 mutation.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3378120
retrieved
17 August 2018
Identifiers
DOI
10.1002/EMMM.200900020
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
Fatcat ID
release_yin3f5k5fjdjni4c7txqrygnjm
1 reference
stated in
Fatcat
reference URL
https://api.fatcat.wiki/v0/release/yin3f5k5fjdjni4c7txqrygnjm
retrieved
24 November 2022
based on heuristic
mapped directly with Wikidata item
PMC publication ID
3378120
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
PubMed publication ID
20049706
1 reference
stated in
Europe PubMed Central
PMC publication ID
3378120
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20049706%20AND%20SRC:MED&resulttype=core&format=json
retrieved
16 January 2020
ResearchGate publication ID
40848572
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