(Q42232540)

19 October 2017

title

MyD88 signalling in myeloid cells is sufficient to prevent chronic mycobacterial infection (English)

1 reference

19 October 2017

author

Catharina Arnold-Schrauf

4

object named as

Catharina Arnold-Schrauf

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19 October 2017

Luciana Berod

1

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19 October 2017

Philipp Stüve

2

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19 October 2017

Maxine Swallow

3

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19 October 2017

Friederike Kruse

5

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19 October 2017

Maria V Gentilini

6

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19 October 2017

Jenny Freitag

7

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19 October 2017

Bernhard Holzmann

8

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19 October 2017

Tim Sparwasser

9

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19 October 2017

language of work or name

English

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publication date

13 February 2014

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European Journal of Immunology

19 October 2017

published in

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19 October 2017

volume

44

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19 October 2017

issue

5

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19 October 2017

page(s)

1399-1409

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How can immunology contribute to the control of tuberculosis?

19 October 2017

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Constrained Intracellular Survival of Mycobacterium tuberculosis in Human Dendritic Cells

21 January 2018

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21 January 2018

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7 January 2021

Bacterial CpG-DNA activates dendritic cells in vivo: T helper cell-independent cytotoxic T cell responses to soluble proteins

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7 January 2021

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7 January 2021

MyD88-mediated stabilization of interferon-gamma-induced cytokine and chemokine mRNA

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7 January 2021

Mycobacterial infection in MyD88-deficient mice

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7 January 2021

IL-1 receptor-mediated signal is an essential component of MyD88-dependent innate response to Mycobacterium tuberculosis infection

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7 January 2021

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7 January 2021

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7 January 2021

Identifiers

DOI

10.1002/EJI.201344039

1 reference

release_jeaupiunqvdmhcqtac4s66o54e

19 October 2017

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/jeaupiunqvdmhcqtac4s66o54e

24 November 2022

mapped directly with Wikidata item

1 reference