(Q42525696)

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Cyclosporine inhibits growth through the activating transcription factor/cAMP-responsive element-binding protein binding site in the cyclin D1 promoter

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Europe PubMed Central

1 November 2017

Cyclosporine inhibits growth through the activating transcription factor/cAMP-responsive element-binding protein binding site in the cyclin D1 promoter (English)

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Europe PubMed Central

1 November 2017

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Florian R Greten

4

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author name string

Gunter Schneider

1

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Europe PubMed Central

1 November 2017

Franz Oswald

2

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Europe PubMed Central

1 November 2017

Christian Wahl

3

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Europe PubMed Central

1 November 2017

Guido Adler

5

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Europe PubMed Central

1 November 2017

Roland M Schmid

6

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Europe PubMed Central

1 November 2017

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publication date

4 September 2002

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Europe PubMed Central

1 November 2017

Journal of Biological Chemistry

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Europe PubMed Central

1 November 2017

277

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Europe PubMed Central

1 November 2017

46

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Europe PubMed Central

1 November 2017

43599-43607

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Europe PubMed Central

1 November 2017

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pp60(v-src) induction of cyclin D1 requires collaborative interactions between the extracellular signal-regulated kinase, p38, and Jun kinase pathways. A role for cAMP response element-binding protein and activating transcription factor-2 in pp60(v-

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Inhibition of cyclic AMP response element-binding protein/cyclic AMP response element-mediated transcription by the immunosuppressive drugs cyclosporin A and FK506 depends on the promoter context.

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Effects of cyclosporin A and a non-immunosuppressive analogue, O-acetyl cyclosporin A, upon the growth of parent and multidrug resistant human lung cancer cells in vitro

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Cyclic 3,5 adenoise monophosphate and cyclosporin A inhibit cellular proliferation and serine/threonine protein phosphatase activity in pituitary cells

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Changes of the trans-activating potential of AP-1 transcription factor during cyclosporin A-induced apoptosis of glioma cells are mediated by phosphorylation and alterations of AP-1 composition

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NF-kappaB controls cell growth and differentiation through transcriptional regulation of cyclin D1.

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Fos family members induce cell cycle entry by activating cyclin D1.

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Cyclin D1, B and A expression and cell turnover in psoriatic skin lesions before and after cyclosporin treatment

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Identifiers

10.1074/JBC.M204787200

1 reference

Europe PubMed Central

1 November 2017

1 reference

Europe PubMed Central

1 November 2017

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