(Q42601896)

3 November 2017

title

Cu2+-induced modification of the kinetics of A beta(1-42) channels (English)

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3 November 2017

5

Colin L Masters

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3 November 2017

Randa Bahadi

1

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3 November 2017

Peter V Farrelly

2

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3 November 2017

Bronwyn L Kenna

3

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3 November 2017

Cyril C Curtain

4

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3 November 2017

Roberto Cappai

6

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3 November 2017

Kevin J Barnham

7

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3 November 2017

Joseph I Kourie

8

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3 November 2017

publication date

18 June 2003

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American Journal of Physiology - Cell Physiology

3 November 2017

published in

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3 November 2017

volume

285

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3 November 2017

issue

4

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3 November 2017

page(s)

C873-80

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beta-Amyloid Ca(2+)-channel hypothesis for neuronal death in Alzheimer disease

3 November 2017

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Zn2+ interaction with Alzheimer amyloid beta protein calcium channels

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Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum

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Dramatic aggregation of Alzheimer abeta by Cu(II) is induced by conditions representing physiological acidosis.

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Characterization of Copper Interactions with Alzheimer Amyloid β Peptides

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Desensitization of mouse nicotinic acetylcholine receptor channels. A two-gate mechanism

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21 January 2018

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Hydrogen peroxide mediates amyloid beta protein toxicity

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21 January 2018

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Rapid induction of Alzheimer A beta amyloid formation by zinc.

21 January 2018

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Treatment with a copper-zinc chelator markedly and rapidly inhibits beta-amyloid accumulation in Alzheimer's disease transgenic mice.

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Metal ions, pH, and cholesterol regulate the interactions of Alzheimer's disease amyloid-beta peptide with membrane lipid

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Alzheimer's disease amyloid-beta binds copper and zinc to generate an allosterically ordered membrane-penetrating structure containing superoxide dismutase-like subunits

21 January 2018

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Both the environment and somatic mutations govern the aggregation pathway of pathogenic immunoglobulin light chain

21 January 2018

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Mechanisms of barbiturate inhibition of acetylcholine receptor channels

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21 January 2018

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Formation of a copper specific binding site in non-native states of beta-2-microglobulin.

21 January 2018

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Pore formation by beta-2-microglobulin: a mechanism for the pathogenesis of dialysis associated amyloidosis

21 January 2018

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21 January 2018

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Copper and zinc binding modulates the aggregation and neurotoxic properties of the prion peptide PrP106-126.

21 January 2018

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Nerve endings from rat brain tissue release copper upon depolarization. A possible role in regulating neuronal excitability

21 January 2018

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Alzheimer's beta-amyloid, human islet amylin, and prion protein fragment evoke intracellular free calcium elevations by a common mechanism in a hypothalamic GnRH neuronal cell line

21 January 2018

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Alzheimer's disease amyloid beta-protein forms Zn(2+)-sensitive, cation-selective channels across excised membrane patches from hypothalamic neurons

21 January 2018

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Cation selective channels formed by a C-terminal fragment of beta-amyloid precursor protein.

21 January 2018

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Mechanisms of amyloid beta protein-induced modification in ion transport systems: implications for neurodegenerative diseases

21 January 2018

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Heterogeneous amyloid-formed ion channels as a common cytotoxic mechanism: implications for therapeutic strategies against amyloidosis

21 January 2018

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Channel activity of deamidated isoforms of prion protein fragment 106-126 in planar lipid bilayers

21 January 2018

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Diversity of amyloid beta protein fragment [1-40]-formed channels.

21 January 2018

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Characteristics of two types of chloride channel in sarcoplasmic reticulum vesicles from rabbit skeletal muscle

21 January 2018

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Amyloid beta protein forms ion channels: implications for Alzheimer's disease pathophysiology

21 January 2018

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Amyloid beta protein (1-40) forms calcium-permeable, Zn2+-sensitive channel in reconstituted lipid vesicles

21 January 2018

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Metal binding modes of Alzheimer's amyloid beta-peptide in insoluble aggregates and soluble complexes.

21 January 2018

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Metalloenzyme-like activity of Alzheimer's disease beta-amyloid. Cu-dependent catalytic conversion of dopamine, cholesterol, and biological reducing agents to neurotoxic H(2)O(2).

21 January 2018

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Measuring kinetics of complex single ion channel data using mean-variance histograms

21 January 2018

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Ion channel hypothesis for Alzheimer amyloid peptide neurotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPCELL.00147.2003

A new hypothesis for the mechanism of amyloid toxicity, based on the calcium channel activity of amyloid beta protein (A beta P) in phospholipid bilayer membranes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPCELL.00147.2003

Amyloid beta protein-(1-42) forms calcium-permeable, Zn2+-sensitive channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPCELL.00147.2003

Aggregates of a beta-amyloid peptide are required to induce calcium currents in neuron-like human teratocarcinoma cells: relation to Alzheimer's disease

21 January 2018

1 reference

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Amyloid beta peptides act directly on single neurons

21 January 2018

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An increased percentage of long amyloid beta protein secreted by familial amyloid beta protein precursor (beta APP717) mutants.

21 January 2018

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Metal-triggered structural transformations, aggregation, and fibrillation of human alpha-synuclein. A possible molecular NK between Parkinson's disease and heavy metal exposure

21 January 2018

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Displacement currents associated with the insertion of Alzheimer disease amyloid beta-peptide into planar bilayer membranes

21 January 2018

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Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides

21 January 2018

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Therapeutic targets in the biology of Alzheimerʼs disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPCELL.00147.2003

10.1152/AJPCELL.00147.2003

21 January 2018

Identifiers

DOI

1 reference

3 November 2017

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