(Q42932553)

English

Insulin Secretory Granules Enter a Highly Calcium‐Sensitive State following Palmitate‐Induced Dissociation from Calcium Channels: A Theoretical Study

scientific article published on December 1, 2010

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Insulin secretory granules enter a highly calcium-sensitive state following palmitate-induced dissociation from calcium channels: a theoretical study (English)

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Insulin Secretory Granules Enter a Highly Calcium‐Sensitive State following Palmitate‐Induced Dissociation from Calcium Channels: A Theoretical Study (English)

1 reference

10.1111/J.1365-2826.2010.02056.X

https://api.crossref.org/works/10.1111/J.1365-2826.2010.02056.X

19 June 2024

Morten Gram Pedersen

1

object named as

M. G. Pedersen

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A20722978

19 June 2024

language of work or name

1 reference

National Center for Biotechnology Information

PubMed publication ID

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=20722978

19 June 2024

publication date

1 December 2010

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

full work available at URL

https://api.wiley.com/onlinelibrary/tdm/v1/articles/10.1111%2Fj.1365-2826.2010.02056.x

1 reference

10.1111/J.1365-2826.2010.02056.X

https://api.crossref.org/works/10.1111/J.1365-2826.2010.02056.X

19 June 2024

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1365-2826.2010.02056.x

1 reference

10.1111/J.1365-2826.2010.02056.X

https://api.crossref.org/works/10.1111/J.1365-2826.2010.02056.X

19 June 2024

Journal of Neuroendocrinology

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

22

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

12

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

1315-1324

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

Phasic and tonic modes of depolarization-exocytosis coupling in beta-cells of porcine islets of Langerhans

1 reference

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Electrical activity and exocytotic correlates of biphasic insulin secretion from beta-cells of canine islets of Langerhans: contribution of tuning two modes of Ca2+ entry-dependent exocytosis to two modes of glucose-induced electrical activity.

1 reference

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Triggering and amplifying pathways of regulation of insulin secretion by glucose

1

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Dynamics of Insulin Secretion by the Perfused Rat Pancreas

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PLASMA-INSULIN RESPONSE TO SUSTAINED HYPERGLYCEMIA INDUCED BY GLUCOSE INFUSION IN HUMAN SUBJECTS

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In vivo and in vitro glucose-induced biphasic insulin secretion in the mouse: pattern and role of cytoplasmic Ca2+ and amplification signals in beta-cells

4

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Phasic insulin release and metabolic regulation in type 2 diabetes.

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Is reduced first-phase insulin release the earliest detectable abnormality in individuals destined to develop type 2 diabetes?

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Fatty acids and insulin secretion

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Long-term exposure to glucose and lipids inhibits glucose-induced insulin secretion downstream of granule fusion with plasma membrane

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Chronic palmitate exposure inhibits insulin secretion by dissociation of Ca(2+) channels from secretory granules

9

1 reference

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Insulin granule dynamics in pancreatic beta cells

10

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TIRF imaging of docking and fusion of single insulin granule motion in primary rat pancreatic beta-cells: different behaviour of granule motion between normal and Goto-Kakizaki diabetic rat beta-cells

11

1 reference

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Imaging analysis reveals mechanistic differences between first- and second-phase insulin exocytosis

12

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Docking is not a prerequisite but a temporal constraint for fusion of secretory granules

13

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Fast exocytosis with few Ca(2+) channels in insulin-secreting mouse pancreatic B cells

14

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Vesicle pools and Ca2+ microdomains: new tools for understanding their roles in neurotransmitter release

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Ca2+ microdomains and the control of insulin secretion

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The voltage sensitive Lc-type Ca2+ channel is functionally coupled to the exocytotic machinery

17

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Protein kinase activation increases insulin secretion by sensitizing the secretory machinery to Ca2+.

18

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A highly Ca2+-sensitive pool of granules is regulated by glucose and protein kinases in insulin-secreting INS-1 cells

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Synaptotagmins bind calcium to release insulin.

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Novel aspects of the molecular mechanisms controlling insulin secretion

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Newcomer insulin secretory granules as a highly calcium-sensitive pool

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Identifying the targets of the amplifying pathway for insulin secretion in pancreatic beta-cells by kinetic modeling of granule exocytosis.

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Modeling buffered Ca2+ diffusion near the membrane: implications for secretion in neuroendocrine cells

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Capacitance measurements of exocytosis in mouse pancreatic alpha-, beta- and delta-cells within intact islets of Langerhans

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SUR1 regulates PKA-independent cAMP-induced granule priming in mouse pancreatic B-cells

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Exocytosis elicited by action potentials and voltage-clamp calcium currents in individual mouse pancreatic B-cells

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Delay between fusion pore opening and peptide release from large dense-core vesicles in neuroendocrine cells

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Dissection of three Ca2+-dependent steps leading to secretion in chromaffin cells from mouse adrenal slices

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CaM kinase II-dependent mobilization of secretory granules underlies acetylcholine-induced stimulation of exocytosis in mouse pancreatic B-cells

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Ca2+ microdomains near plasma membrane Ca2+ channels: impact on cell function

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Interactions of presynaptic Ca2+ channels and snare proteins in neurotransmitter release

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Syntaxin 1 interacts with the L(D) subtype of voltage-gated Ca(2+) channels in pancreatic beta cells

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Antibody inhibition of synaptosomal protein of 25 kDa (SNAP-25) and syntaxin 1 reduces rapid exocytosis in insulin-secreting cells

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Identifiers

10.1111/J.1365-2826.2010.02056.X

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

11 November 2017

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