(Q43691707)

25 November 2017

Statistical aspects of quantitative image analysis of beta-amyloid in the APP(V717F) transgenic mouse model of Alzheimer's disease (English)

1 reference

25 November 2017

0 references

1 reference

0 references

Fishman CE

1

1 reference

25 November 2017

Cummins DJ

2

1 reference

25 November 2017

Bales KR

3

1 reference

25 November 2017

DeLong CA

4

1 reference

25 November 2017

Esterman MA

5

1 reference

25 November 2017

Hanson JC

6

1 reference

25 November 2017

White SL

7

1 reference

25 November 2017

Paul SM

8

1 reference

25 November 2017

Jordan WH

9

1 reference

25 November 2017

1 July 2001

1 reference

Journal of Neuroscience Methods

25 November 2017

1 reference

25 November 2017

108

1 reference

25 November 2017

2

1 reference

25 November 2017

145-152

1 reference

Apolipoprotein E is essential for amyloid deposition in the APP(V717F) transgenic mouse model of Alzheimer's disease

25 November 2017

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Lack of apolipoprotein E dramatically reduces amyloid beta-peptide deposition.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Accelerated amyloid deposition in the brains of transgenic mice coexpressing mutant presenilin 1 and amyloid precursor proteins.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Neuron loss in APP transgenic mice

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Aging increased amyloid peptide and caused amyloid plaques in brain of old APP/V717I transgenic mice by a different mechanism than mutant presenilin1

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Prospects for noninvasive imaging of brain amyloid beta in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

High-throughput quantitative histological analysis of Alzheimer's disease pathology using a confocal digital microscanner

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Amyloid, the presenilins and Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Alzheimer's disease: the amyloid cascade hypothesis

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

APPSw transgenic mice develop age-related A beta deposits and neuropil abnormalities, but no neuronal loss in CA1.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Abeta deposition is associated with neuropil changes, but not with overt neuronal loss in the human amyloid precursor protein V717F (PDAPP) transgenic mouse.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Ibuprofen suppresses plaque pathology and inflammation in a mouse model for Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Comparison of pathological diagnostic criteria for Alzheimer disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Brain trauma in aged transgenic mice induces regression of established abeta deposits

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Hypercholesterolemia accelerates the Alzheimer's amyloid pathology in a transgenic mouse model

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Alzheimer's disease: a central role for amyloid

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

In vivo detection of amyloid plaques in a mouse model of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

Amyloidogenic role of cytokine TGF-beta1 in transgenic mice and in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0165-0270%2801%2900381-8

7 January 2021

10.1016/S0165-0270(01)00381-8

Identifiers

DOI

1 reference

25 November 2017

1 reference