(Q44062492)

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Sophoranone, extracted from a traditional Chinese medicine Shan Dou Gen, induces apoptosis in human leukemia U937 cells via formation of reactive oxygen species and opening of mitochondrial permeability transition pores

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scholarly article

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Europe PubMed Central

29 November 2017

Sophoranone, extracted from a traditional Chinese medicine Shan Dou Gen, induces apoptosis in human leukemia U937 cells via formation of reactive oxygen species and opening of mitochondrial permeability transition pores (English)

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Europe PubMed Central

29 November 2017

2 references

ChEBI release 2020-05-28

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inferred from title

reactive oxygen species

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inferred from title

Sophora tonkinensis

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traditional Chinese medicine

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object named as

traditional Chinese medicine

17 February 2024

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Sachiko Kajimoto

1

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Europe PubMed Central

29 November 2017

Noriko Takanashi

2

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Europe PubMed Central

29 November 2017

Tetsuya Kajimoto

3

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Europe PubMed Central

29 November 2017

Man Xu

4

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Europe PubMed Central

29 November 2017

Judong Cao

5

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Europe PubMed Central

29 November 2017

Yutaka Masuda

6

1 reference

Europe PubMed Central

29 November 2017

Toshihiro Aiuchi

7

1 reference

Europe PubMed Central

29 November 2017

Shigeo Nakajo

8

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Europe PubMed Central

29 November 2017

Yoshiteru Ida

9

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Europe PubMed Central

29 November 2017

Kazuyasu Nakaya

10

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29 November 2017

language of work or name

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publication date

1 June 2002

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29 November 2017

full work available at URL

https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/ijc.10414

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19 March 2024

https://api.unpaywall.org/v2/10.1002/ijc.10414?email=unpaywall_01@example.com

International Journal of Cancer

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29 November 2017

99

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29 November 2017

879-890

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29 November 2017

6

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29 November 2017

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19 March 2024

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Activation of programmed cell death (apoptosis) by cisplatin, other anticancer drugs, toxins and hyperthermia

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The cooperative interaction of two different signaling pathways in response to bufalin induces apoptosis in human leukemia U937 cells

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Citrus flavone tangeretin inhibits leukaemic HL-60 cell growth partially through induction of apoptosis with less cytotoxicity on normal lymphocytes

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Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.

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Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

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Activation of the apoptotic protease CPP32 by cytotoxic T-cell-derived granzyme B.

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Evidence for enhanced vascular superoxide anion production in nitrate tolerance. A novel mechanism underlying tolerance and cross-tolerance

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Involvement of reactive oxygen species and caspase 3 activation in arsenite-induced apoptosis

1 reference

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Arsenic trioxide-induced apoptosis and differentiation are associated respectively with mitochondrial transmembrane potential collapse and retinoic acid signaling pathways in acute promyelocytic leukemia

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Release of cytochrome c from liver mitochondria during permeability transition.

1 reference

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Daidzein inhibits insulin- or insulin-like growth factor-1-mediated signaling in cell cycle progression of Swiss 3T3 cells

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1 reference

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Growth inhibition with reversible cell cycle arrest of carcinoma cells by flavone L86-8275

1 reference

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release

1 reference

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity

1 reference

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Absence of cardiolipin in the crd1 null mutant results in decreased mitochondrial membrane potential and reduced mitochondrial function

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Implication of mitochondria-derived reactive oxygen species, cytochrome C and caspase-3 in N-(4-hydroxyphenyl)retinamide-induced apoptosis in cervical carcinoma cells.

1 reference

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Mechanism of vanadate-induced activation of tyrosine phosphorylation and of the respiratory burst in HL60 cells. Role of reduced oxygen metabolites

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Pro-inflammatory cytokines and environmental stress cause p38 mitogen-activated protein kinase activation by dual phosphorylation on tyrosine and threonine

1 reference

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Signal transduction by tumor necrosis factor mediated by JNK protein kinases

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Cisplatin induces a persistent activation of JNK that is related to cell death

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Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis.

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Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

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Stress-signalling kinase Sek1 protects thymocytes from apoptosis mediated by CD95 and CD3

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Early activation of c-Jun N-terminal kinase and p38 kinase regulate cell survival in response to tumor necrosis factor alpha

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Oxygen-derived free radicals in postischemic tissue injury

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Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells

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The Activation of the c-Jun N-terminal Kinase and p38 Mitogen-activated Protein Kinase Signaling Pathways Protects HeLa Cells from Apoptosis Following Photodynamic Therapy with Hypericin

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Topoisomerase inhibitors induce apoptosis in thymocytes

1 reference

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Geranylgeraniol is a potent inducer of apoptosis in tumor cells

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beta-hydroxyisovalerylshikonin inhibits the cell growth of various cancer cell lines and induces apoptosis in leukemia HL-60 cells through a mechanism different from those of Fas and etoposide

1 reference

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Induction of apoptosis by bufalin in human tumor cells is associated with a change of intracellular concentration of Na+ ions

1 reference

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Activation of AP-1 is required for bufalin-induced apoptosis in human leukemia U937 cells

1 reference

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Implication of mitochondrial hydrogen peroxide generation in ceramide-induced apoptosis

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Geranylgeranylacetone used as an antiulcer agent is a potent inducer of differentiation of various human myeloid leukemia cell lines

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Studies on the Constituents of Sophora Species. I. Constituents of Sophora subprostrata CHUN et T. CHEN. (1). Isolation and Structure of New Flavonoids, Sophoradin and Sophoranone

1 reference

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10.1002/IJC.10414

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Europe PubMed Central

29 November 2017

release_ey55x5jqtbaebgtx74omp5tv2y

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https://api.fatcat.wiki/v0/release/ey55x5jqtbaebgtx74omp5tv2y

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29 November 2017

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