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Acute and chronic alterations in calcium homeostasis in 3-nitropropionic acid-treated human NT2-N neurons.
scientific article published in January 2002
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scholarly article
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Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
title
Acute and chronic alterations in calcium homeostasis in 3-nitropropionic acid-treated human NT2-N neurons
(English)
1 reference
stated in
Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
author
Wang-Tso Lee
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
author name string
T Itoh
series ordinal
2
1 reference
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Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
D Pleasure
series ordinal
3
1 reference
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Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
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11 November 2019
language of work or name
English
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publication date
1 January 2002
1 reference
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Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
published in
Neuroscience
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Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
volume
113
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stated in
Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
issue
3
1 reference
stated in
Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
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11 November 2019
page(s)
699-708
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Europe PubMed Central
PubMed publication ID
12150790
reference URL
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11 November 2019
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Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid
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Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases
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Exacerbated responses to oxidative stress by an Na(+) load in isolated nerve terminals: the role of ATP depletion and rise of [Ca(2+)](i).
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Energy and glutamate dependency of 3-Nitropropionic acid neurotoxicity in culture
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Effect of exogenous and endogenous antioxidants on 3-nitropionic acid-induced in vivo oxidative stress and striatal lesions: insights into Huntington's disease
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Cyclosporin A, but not FK 506, protects mitochondria and neurons against hypoglycemic damage and implicates the mitochondrial permeability transition in cell death.
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Astrocytes are more vulnerable than neurons to cellular Ca2+ overload induced by a mitochondrial toxin, 3-nitropropionic acid
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3-Nitropropionic acid exacerbates N-methyl-D-aspartate toxicity in striatal culture by multiple mechanisms.
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AMPA receptor-mediated excitotoxicity in human NT2-N neurons results from loss of intracellular Ca2+ homeostasis following marked elevation of intracellular Na+.
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o-Phthaldialdehyde precolumn derivatization and reversed-phase high-performance liquid chromatography of polypeptide hydrolysates and physiological fluids
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Increased sensitivity to mitochondrial toxin-induced apoptosis in neural cells expressing mutant presenilin-1 is linked to perturbed calcium homeostasis and enhanced oxyradical production.
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Blockade of mitochondrial Ca2+ uptake by mitochondrial inhibitors amplifies the glutamate-induced calcium response in cultured cerebellar granule cells.
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Excitotoxicity is required for induction of oxidative stress and apoptosis in mouse striatum by the mitochondrial toxin, 3-nitropropionic acid
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Mice deficient in cellular glutathione peroxidase show increased vulnerability to malonate, 3-nitropropionic acid, and 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridine
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Neuroprotective effect of lamotrigine and MK-801 on rat brain lesions induced by 3-nitropropionic acid: evaluation by magnetic resonance imaging and in vivo proton magnetic resonance spectroscopy
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Cyclosporin A protects striatal neurons in vitro and in vivo from 3-nitropropionic acid toxicity.
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Crossref
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Amelioration by cyclosporin A of brain damage following 5 or 10 min of ischemia in rats subjected to preischemic hyperglycemia.
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Compromised mitochondrial function leads to increased cytosolic calcium and to activation of MAP kinases
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7 January 2021
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Presenilin-1 mutation increases neuronal vulnerability to focal ischemia in vivo and to hypoxia and glucose deprivation in cell culture: involvement of perturbed calcium homeostasis
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NMDA receptor-dependent excitotoxicity: the role of intracellular Ca2+ release
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3-Nitropropionic acid: an astrocyte-sparing neurotoxin in vitro
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Crossref
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Chronic 3-nitropropionic acid treatment in baboons replicates the cognitive and motor deficits of Huntington's disease
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Crossref
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Mechanisms of cell death induced by the mitochondrial toxin 3-nitropropionic acid: acute excitotoxic necrosis and delayed apoptosis.
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Pure, postmitotic, polarized human neurons derived from NTera 2 cells provide a system for expressing exogenous proteins in terminally differentiated neurons
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1 reference
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Crossref
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7 January 2021
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inferred from DOI database lookup
Hypoxic cell death in human NT2-N neurons: involvement of NMDA and non-NMDA glutamate receptors.
1 reference
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Crossref
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7 January 2021
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inferred from DOI database lookup
Involvement of oxidative stress in 3-nitropropionic acid neurotoxicity
1 reference
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Crossref
reference URL
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Modulation of Bcl-2 expression: a potential component of estrogen protection in NT2 neurons
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Crossref
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7 January 2021
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Glutamate-induced neuron death requires mitochondrial calcium uptake
1 reference
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Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
3‐Nitropropionic Acid Toxicity in the Striatum
1 reference
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Crossref
reference URL
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inferred from DOI database lookup
Inducible expression of neuronal glutamate receptor channels in the NT2 human cell line
1 reference
stated in
Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
The endoplasmic reticulum stress-responsive protein GRP78 protects neurons against excitotoxicity and apoptosis: suppression of oxidative stress and stabilization of calcium homeostasis
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900177-X
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identifiers
DOI
10.1016/S0306-4522(02)00177-X
1 reference
stated in
Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
PubMed publication ID
12150790
1 reference
stated in
Europe PubMed Central
PubMed publication ID
12150790
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12150790%20AND%20SRC:MED&resulttype=core&format=json
retrieved
11 November 2019
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