(Q44605170)

English

Enhanced Akt signaling is an early pro-survival response that reflects N-methyl-D-aspartate receptor activation in Huntington's disease knock-in striatal cells

scientific article published in October 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Enhanced Akt signaling is an early pro-survival response that reflects N-methyl-D-aspartate receptor activation in Huntington's disease knock-in striatal cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Huntington's disease

0 references

1 reference

based on heuristic

inferred from title

Carlos A. Saura

4

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Marcy E. MacDonald

5

object named as

Marcy E MacDonald

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

author name string

Silvia Gines

1

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Elena Ivanova

2

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Ihn-Sik Seong

3

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

language of work or name

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publication date

1 October 2003

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

4 December 2017

278

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Europe PubMed Central

PubMed publication ID

4 December 2017

50

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

50514-50522

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Huntington disease.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

CAG repeat number governs the development rate of pathology in Huntington's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Neurological abnormalities in a knock-in mouse model of Huntington's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Decrease in striatal enkephalin mRNA in mouse models of Huntington's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Specific progressive cAMP reduction implicates energy deficit in presymptomatic Huntington's disease knock-in mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Identification of a presymptomatic molecular phenotype in Hdh CAG knock-in mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Long glutamine tracts cause nuclear localization of a novel form of huntingtin in medium spiny striatal neurons in HdhQ92 and HdhQ111 knock-in mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Early phenotypes that presage late-onset neurodegenerative disease allow testing of modifiers in Hdh CAG knock-in mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Loss of huntingtin-mediated BDNF gene transcription in Huntington's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Dominant phenotypes produced by the HD mutation in STHdh(Q111) striatal cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

In vitro effects of polyglutamine tracts on Ca2+-dependent depolarization of rat and human mitochondria: relevance to Huntington's disease.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Transcription-dependent and -independent control of neuronal survival by the PI3K-Akt signaling pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Activity-dependent NMDA receptor-mediated activation of protein kinase B/Akt in cortical neuronal cultures

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

The IGF-1/Akt pathway is neuroprotective in Huntington's disease and involves Huntingtin phosphorylation by Akt.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Cellular survival: a play in three Akts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Destabilization of beta-catenin by mutations in presenilin-1 potentiates neuronal apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

NMDA receptor function in mouse models of Huntington disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Alternative excitotoxic hypotheses

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Bioenergetics and glutamate excitotoxicity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Mitochondrial dysfunction in neurodegenerative diseases

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Polyglutamine-expanded huntingtin promotes sensitization of N-methyl-D-aspartate receptors via post-synaptic density 95.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Huntingtin and huntingtin-associated protein 1 influence neuronal calcium signaling mediated by inositol-(1,4,5) triphosphate receptor type 1

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M309348200

21 January 2018

Identifiers

10.1074/JBC.M309348200

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

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