(Q45876208)

17 December 2017

title

Round two for liver gene therapy (English)

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17 December 2017

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Wilson JM

1

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17 December 2017

language of work or name

English

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publication date

1 March 1996

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17 December 2017

published in

Nature Genetics

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17 December 2017

volume

12

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17 December 2017

issue

3

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17 December 2017

page(s)

232-233

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17 December 2017

https://scigraph.springernature.com/pub.10.1038/ng0396-232

exact match

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cites work

Hepatocytes corrected by gene therapy are selected in vivo in a murine model of hereditary tyrosinaemia type I.

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

A pilot study of ex vivo gene therapy for homozygous familial hypercholesterolaemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

Pharmacological correction of neonatal lethal hepatic dysfunction in a murine model of hereditary tyrosinaemia type I.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

Long-Term Improvement of Hypercholesterolemia After Ex Vivo Gene Therapy in LDLR-Deficient Rabbits

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Successful ex vivo gene therapy directed to liver in a patient with familial hypercholesterolaemia

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Hereditary tyrosinemia type I. Self-induced correction of the fumarylacetoacetase defect

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Self-induced correction of the genetic defect in tyrosinemia type I

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Complete hepatic regeneration after somatic deletion of an albumin-plasminogen activator transgene

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Replacement of diseased mouse liver by hepatic cell transplantation

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Loss of fumarylacetoacetate hydrolase is responsible for the neonatal hepatic dysfunction phenotype of lethal albino mice.

1 reference

https://api.crossref.org/works/10.1038%2FNG0396-232

7 January 2021

Identifiers

DOI

10.1038/NG0396-232

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17 December 2017

Dimensions Publication ID

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1 reference

17 December 2017