(Q46487407)

English

Abeta(31-35) and Abeta(25-35) fragments of amyloid beta-protein induce cellular death through apoptotic signals: Role of the redox state of methionine-35.

scientific article published in May 2005

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scholarly article

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11 January 2020

Abeta(31-35) and Abeta(25-35) fragments of amyloid beta-protein induce cellular death through apoptotic signals: Role of the redox state of methionine-35 (English)

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11 January 2020

apoptotic process

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Maria Elisabetta Clementi

1

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11 January 2020

Francesco Misiti

6

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11 January 2020

5

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Bruno Giardina

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11 January 2020

Massimo Coletta

3

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Massimo Coletta

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11 January 2020

author name string

Stefano Marini

2

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11 January 2020

Federica Orsini

4

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11 January 2020

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1 May 2005

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11 January 2020

1 reference

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11 January 2020

579

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11 January 2020

13

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11 January 2020

2913-2918

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11 January 2020

X-ray diffraction from intraneuronal paired helical filaments and extraneuronal amyloid fibers in Alzheimer disease indicates cross-beta conformation

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

In vivo conversion of racemized beta-amyloid ([D-Ser 26]A beta 1-40) to truncated and toxic fragments ([D-Ser 26]A beta 25-35/40) and fragment presence in the brains of Alzheimer's patients

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Amyloid, the presenilins and Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Exploring the etiology of Alzheimer disease using molecular genetics

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Effects of beta-amyloid peptides on the fluidity of membranes from frontal and parietal lobes of human brain. High potencies of A beta 1-42 and A beta 1-43.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Amyloid beta protein-(1-42) forms calcium-permeable, Zn2+-sensitive channel

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Oligomeric and fibrillar species of amyloid-beta peptides differentially affect neuronal viability.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Amyloid beta peptide induces cytochrome C release from isolated mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Apoptosis is induced by beta-amyloid in cultured central nervous system neurons

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Caspases: killer proteases

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Beta-amyloid peptide fragment 31-35 induces apoptosis in cultured cortical neurons.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Methionine residue 35 is critical for the oxidative stress and neurotoxic properties of Alzheimer's amyloid beta-peptide 1-42.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Different mechanisms of oxidative stress and neurotoxicity for Alzheimer's A beta(1--42) and A beta(25--35).

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Comparative analysis of amyloid-beta chemical structure and amyloid plaque morphology of transgenic mouse and Alzheimer's disease brains

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Oxidation of methionyl residues in proteins: tools, targets, and reversal

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

The serpin-enzyme complex receptor recognizes soluble, nontoxic amyloid-beta peptide but not aggregated, cytotoxic amyloid-beta peptide

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Physical, morphological and functional differences between ph 5.8 and 7.4 aggregates of the Alzheimer's amyloid peptide Abeta

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Preparation of synaptic and nonsynaptic mitochondria from mammalian brain

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Methionine 35 oxidation reduces toxic effects of the amyloid beta-protein fragment (31-35) on human red blood cell

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Methionine 35 oxidation reduces toxic and pro-apoptotic effects of the amyloid beta-protein fragment (31-35) on isolated brain mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Amyloid beta-protein aggregation nullifies its pathologic properties in cultured cerebrovascular smooth muscle cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

beta-Amyloid fragment 25-35 selectively decreases complex IV activity in isolated mitochondria.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Modulation of the mitochondrial permeability transition pore by pyridine nucleotides and dithiol oxidation at two separate sites.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Effects of curcumin and curcumin derivatives on mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Heparan sulfate and chondroitin sulfate glycosaminoglycan attenuate beta-amyloid(25-35) induced neurodegeneration in cultured hippocampal neurons.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Abeta25-35 induces rapid lysis of red blood cells: contrast with Abeta1-42 and examination of underlying mechanisms

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Substitution of isoleucine-31 by helical-breaking proline abolishes oxidative stress and neurotoxic properties of Alzheimer's amyloid beta-peptide

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Comparing the effect on protein stability of methionine oxidation versus mutagenesis: steps toward engineering oxidative resistance in proteins

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Neurotoxic, redox-competent Alzheimer's beta-amyloid is released from lipid membrane by methionine oxidation

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

Decrease in peptide methionine sulfoxide reductase in Alzheimer's disease brain.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2005.04.041

21 January 2018

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