(Q46939197)

26 December 2017

title

Increased mitochondrial calcium coexists with decreased reperfusion injury in postconditioned (but not preconditioned) hearts (English)

1 reference

26 December 2017

1 reference

5

Joseph Loufouat

1 reference

26 December 2017

Michel Ovize

8

Michel Ovize

1 reference

26 December 2017

Laurent Argaud

1

Laurent Argaud

1 reference

26 December 2017

Odile Gateau-Roesch

2

Odile Gateau-Roesch

1 reference

26 December 2017

Lionel Augeul

3

Lionel Augeul

1 reference

26 December 2017

Elisabeth Couture-Lepetit

4

Elisabeth Couture-Lepetit

1 reference

26 December 2017

Ludovic Gomez

6

Ludovic Gomez

1 reference

26 December 2017

author name string

Dominique Robert

7

1 reference

26 December 2017

publication date

19 October 2007

1 reference

26 December 2017

American Journal of Physiology Heart and Circulatory Physiology

published in

1 reference

26 December 2017

volume

294

1 reference

26 December 2017

issue

1

1 reference

26 December 2017

page(s)

H386-91

1 reference

26 December 2017

Ischemic and anesthetic preconditioning reduces cytosolic [Ca2+] and improves Ca(2+) responses in intact hearts

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Preconditioning delays Ca2+-induced mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Specific inhibition of the mitochondrial permeability transition prevents lethal reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Ceramide in the antiapoptotic effect of ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

PI 3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Low-pressure reperfusion alters mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Isoflurane protects against myocardial infarction during early reperfusion by activation of phosphatidylinositol-3-kinase signal transduction: evidence for anesthetic-induced postconditioning in rabbits

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mitochondrial intermembrane junctional complexes and their role in cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

On the involvement of mitochondrial intermembrane junctional complexes in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Isoflurane postconditioning prevents opening of the mitochondrial permeability transition pore through inhibition of glycogen synthase kinase 3beta.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Cyclophilin and peptidyl-prolyl cis-trans isomerase are probably identical proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Fas-independent mitochondrial damage triggers cardiomyocyte death after ischemia-reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mechanisms by which mitochondria transport calcium

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Regulated and unregulated mitochondrial permeability transition pores: a new paradigm of pore structure and function?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mitochondrial permeability transition: a common pathway to necrosis and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Postconditioning attenuates myocardial ischemia-reperfusion injury by inhibiting events in the early minutes of reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mitochondrial targeted cyclophilin D protects cells from cell death by peptidyl prolyl isomerization

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Attenuation of postischemic reperfusion injury is related to prevention of [Ca2+]m overload in rat hearts

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mitochondria as all-round players of the calcium game

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Defective calcium handling in cardiomyocytes isolated from hearts subjected to ischemia-reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Cyclophilin D, a component of the permeability transition-pore, is an apoptosis repressor

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Mechanism of preconditioning. Ionic alterations

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Postconditioning: a form of "modified reperfusion" protects the myocardium by activating the phosphatidylinositol 3-kinase-Akt pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Inhibition of the mitochondrial permeability transition by the nonimmunosuppressive cyclosporin derivative NIM811.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Preconditioning limits mitochondrial Ca(2+) during ischemia in rat hearts: role of K(ATP) channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Role of the mitochondrial permeability transition in myocardial disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Postconditioning's protection is not dependent on circulating blood factors or cells but involves adenosine receptors and requires PI3-kinase and guanylyl cyclase activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Infarct-sparing effect of myocardial postconditioning is dependent on protein kinase C signalling

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01035.2007

10.1152/AJPHEART.01035.2007

21 January 2018

Identifiers

DOI

1 reference

26 December 2017

release_wyukrdjrfbcvrgiwl7yxp2opze

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/wyukrdjrfbcvrgiwl7yxp2opze

mapped directly with Wikidata item

24 November 2022

based on heuristic

PubMed ID

17951363

1 reference

26 December 2017