(Q47577090)

4 February 2018

title

Correlation between cognitive deficits and Abeta deposits in transgenic APP+PS1 mice. (English)

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4 February 2018

author name string

Gordon MN

1

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4 February 2018

King DL

2

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4 February 2018

Diamond DM

3

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4 February 2018

Jantzen PT

4

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4 February 2018

Boyett KV

5

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4 February 2018

Hope CE

6

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4 February 2018

Hatcher JM

7

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4 February 2018

DiCarlo G

8

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4 February 2018

Gottschall WP

9

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4 February 2018

Morgan D

10

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4 February 2018

Arendash GW

11

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4 February 2018

publication date

1 May 2001

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4 February 2018

published in

Neurobiology of Aging

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4 February 2018

volume

22

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4 February 2018

page(s)

377-385

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4 February 2018

issue

3

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4 February 2018

Progressive, age-related behavioral impairments in transgenic mice carrying both mutant amyloid precursor protein and presenilin-1 transgenes.

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Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid beta-protein in both transfected cells and transgenic mice

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Tau, paired helical filaments and amyloid in the neocortex: a morphometric study of 15 cases with graded intellectual status in aging and senile dementia of Alzheimer type

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Subtypes and differential laminar distributions of ?A4 deposits in Alzheimer's disease: relationship with the intellectual status of 26 cases

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Visual-discrimination learning ability and beta-amyloid accumulation in the dog.

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Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes

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7 January 2021

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7 January 2021

Progressive and gender-dependent cognitive impairment in the APP(SW) transgenic mouse model for Alzheimer's disease.

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7 January 2021

Reversal of beta-amyloid-induced retention deficit after exposure to training and state cues.

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7 January 2021

Comparison of neurodegenerative pathology in transgenic mice overexpressing V717F beta-amyloid precursor protein and Alzheimer's disease

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7 January 2021

Amnesia induced in mice by centrally administered beta-amyloid peptides involves cholinergic dysfunction.

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7 January 2021

Amyloid phenotype characterization of transgenic mice overexpressing both mutant amyloid precursor protein and mutant presenilin 1 transgenes.

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment

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7 January 2021

Reorganization of cholinergic terminals in the cerebral cortex and hippocampus in transgenic mice carrying mutated presenilin-1 and amyloid precursor protein transgenes.

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7 January 2021

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7 January 2021

10.1016/S0197-4580(00)00249-9

Identifiers

DOI

1 reference

4 February 2018

PubMed ID

11378242

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4 February 2018