(Q47744223)

English

Loss of inositol 1,4,5-trisphosphate receptor sites and decreased PKC levels correlate with staging of Alzheimer's disease neurofibrillary pathology.

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scholarly article

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Loss of inositol 1,4,5-trisphosphate receptor sites and decreased PKC levels correlate with staging of Alzheimer's disease neurofibrillary pathology. (English)

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Alzheimer's disease

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5

object named as

Winblad B

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

author name string

Kurumatani T

1

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Fastbom J

2

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Bonkale WL

3

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Bogdanovic N

4

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Ohm TG

6

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Cowburn RF

7

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5 February 2018

http://europepmc.org/abstract/MED/9689471

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publication date

1 June 1998

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

796

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

1-2

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5 February 2018

http://europepmc.org/abstract/MED/9689471

209-221

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

Acute rise in the concentration of free cytoplasmic calcium leads to dephosphorylation of the microtubule-associated protein tau.

1 reference

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Neuropathological stageing of Alzheimer-related changes

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Processing of Alzheimer beta/A4 amyloid precursor protein: modulation by agents that regulate protein phosphorylation

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Calcium regulates processing of the Alzheimer amyloid protein precursor in a protein kinase C-independent manner

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Protein phosphorylation regulates secretion of Alzheimer beta/A4 amyloid precursor protein

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Decreased levels of protein kinase C in Alzheimer brain

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Cholinergic and serotonergic stimulation of phosphoinositide hydrolysis is decreased in Alzheimer's disease.

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Phospholipase A2 activation influences the processing and secretion of the amyloid precursor protein

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Diminished muscarinic receptor-stimulated [3H]-PIP2 hydrolysis in Alzheimer's disease

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Modulation of the phosphorylation state of tau in situ: the roles of calcium and cyclic AMP.

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Loss of high-affinity agonist binding to M1 muscarinic receptors in Alzheimer's disease: implications for the failure of cholinergic replacement therapies.

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Diminished [3H]inositol(1,4,5)P3 but not [3H]inositol(1,3,4,5)P4 binding in Alzheimer's disease brain.

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Quantitative film autoradiography for tritium: methodological considerations.

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Dephosphorylation of Alzheimer's disease abnormally phosphorylated tau by protein phosphatase-2A.

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Phosphatase activity toward abnormally phosphorylated tau: decrease in Alzheimer disease brain.

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Phosphoinositide hydrolysis, G alpha q, phospholipase C, and protein kinase C in post mortem human brain: effects of post mortem interval, subject age, and Alzheimer's disease.

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Active site-directed antibodies identify calpain II as an early-appearing and pervasive component of neurofibrillary pathology in Alzheimer's disease.

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7 January 2021

Decreased inositol (1,4,5)-trisphosphate receptor levels in Alzheimer's disease cerebral cortex: selectivity of changes and possible correlation to pathological severity.

1 reference

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7 January 2021

Autoradiographic imaging of [3H]phorbol 12,13-dibutyrate binding to protein kinase C in Alzheimer's disease

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Impaired phosphoinositide hydrolysis in Alzheimer's disease brain.

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7 January 2021

Diagnosis of Alzheimer's disease

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Autoradiographic characterization of [3H]inositol (1,4,5) trisphosphate and [3H]inositol (1,3,4,5) tetrakisphosphate binding sites in human brain

1 reference

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7 January 2021

Regional alterations in M1 muscarinic receptor-G protein coupling in Alzheimer's disease.

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Alzheimer's disease-like phosphorylation of the microtubule-associated protein tau by glycogen synthase kinase-3 in transfected mammalian cells.

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Differential involvement of protein kinase C isozymes in Alzheimer's disease

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Protein kinase C alteration is an early biochemical marker in Alzheimer's disease

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7 January 2021

Ca2+-dependent and Ca2+-independent protein kinase C changes in the brain of patients with Alzheimer's disease

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7 January 2021

Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

1 reference

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7 January 2021

The neuroanatomy of Alzheimer's disease

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Calcium ionophore increases amyloid beta peptide production by cultured cells

1 reference

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7 January 2021

Assessment of protein kinase C isozymes by two-site enzyme immunoassay in human brains and changes in Alzheimer's disease.

1 reference

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Guanine nucleotide modulation of muscarinic cholinergic receptor binding in postmortem human brain--a preliminary study in Alzheimer's disease.

1 reference

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7 January 2021

Attenuated protein kinase C activity and translocation in Alzheimer's disease brain.

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Muscarinic regulation of Alzheimer's disease amyloid precursor protein secretion and amyloid beta-protein production in human neuronal NT2N cells

1 reference

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7 January 2021

Decreased brain [3H]inositol 1,4,5-trisphosphate binding in Alzheimer's disease.

1 reference

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7 January 2021

The neurochemistry of Alzheimer's disease.

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Calcium-activated neutral proteinase (calpain) system in aging and Alzheimer's disease

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Distribution, levels, and activity of glycogen synthase kinase-3 in the Alzheimer disease brain.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2898%2900347-3

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1016/S0006-8993(98)00347-3

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/9689471

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