(Q49027151)

17 February 2018

title

A Bcl-2 transgene expressed in hepatocytes does not protect mice from fulminant liver destruction induced by Fas ligand. (English)

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17 February 2018

Peter Vandenabeele

7

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Xavier Saelens

6

object named as

Xavier Saelens

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17 February 2018

author name string

Geert van Loo

1

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17 February 2018

Saskia Lippens

2

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17 February 2018

Michel Hahne

3

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17 February 2018

Filip Matthijssens

4

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17 February 2018

Wim Declercq

5

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17 February 2018

publication date

1 May 2003

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17 February 2018

published in

Cytokine

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17 February 2018

volume

22

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17 February 2018

issue

3-4

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17 February 2018

page(s)

62-70

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17 February 2018

Fas ligand-induced apoptosis

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1 reference

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7 January 2021

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

1 reference

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7 January 2021

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

1 reference

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7 January 2021

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

1 reference

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7 January 2021

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

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7 January 2021

FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).

1 reference

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7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

1 reference

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

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7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

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7 January 2021

Apaf-1 oligomerizes into biologically active approximately 700-kDa and inactive approximately 1.4-MDa apoptosome complexes

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7 January 2021

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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7 January 2021

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

1 reference

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7 January 2021

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)

1 reference

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7 January 2021

Bcl–2 protects from lethal hepatic apoptosis induced by an ant–Fas antibody in mice

1 reference

https://api.crossref.org/works/10.1016%2FS1043-4666%2803%2900111-X

7 January 2021

A bcl-2 transgene expressed in hepatocytes protects mice from fulminant liver destruction but not from rapid death induced by anti-Fas antibody injection

1 reference

https://api.crossref.org/works/10.1016%2FS1043-4666%2803%2900111-X

7 January 2021

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

1 reference

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7 January 2021

Targeted mutation in the Fas gene causes hyperplasia in peripheral lymphoid organs and liver

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7 January 2021

Conversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity

1 reference

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7 January 2021

Characterization of Fas (Apo-1, CD95)-Fas ligand interaction

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7 January 2021

Lethal effect of the anti-Fas antibody in mice

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7 January 2021

Caspase-independent programmed cell death with necrotic morphology

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7 January 2021

Conversion of Bcl-2 to a Bax-like death effector by caspases

1 reference

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7 January 2021

Cross-talk in cell death signaling

1 reference

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7 January 2021

A caspase-activated factor (CAF) induces mitochondrial membrane depolarization and cytochrome c release by a nonproteolytic mechanism.

1 reference

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7 January 2021

Caspase-independent cell killing by Fas-associated protein with death domain

1 reference

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7 January 2021

Necrotic death pathway in Fas receptor signaling

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7 January 2021

Cleavage of BID during cytotoxic drug and UV radiation-induced apoptosis occurs downstream of the point of Bcl-2 action and is catalysed by caspase-3: a potential feedback loop for amplification of apoptosis-associated mitochondrial cytochrome c rel

1 reference

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7 January 2021

Selective localization of Bcl-2 to the inner mitochondrial and smooth endoplasmic reticulum membranes in mammalian cells

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7 January 2021

A matrix-assisted laser desorption ionization post-source decay (MALDI-PSD) analysis of proteins released from isolated liver mitochondria treated with recombinant truncated Bid.

1 reference

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7 January 2021

The proteolytic procaspase activation network: an in vitro analysis.

1 reference

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7 January 2021

An improved colorimetric assay for interleukin 2.

1 reference

https://api.crossref.org/works/10.1016%2FS1043-4666%2803%2900111-X

7 January 2021

10.1016/S1043-4666(03)00111-X

Identifiers

DOI

1 reference

17 February 2018

1 reference

17 February 2018