(Q50736052)

18 March 2018

title

The stability and anti-apoptotic function of A1 are controlled by its C terminus. (English)

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18 March 2018

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Marco J Herold

1

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18 March 2018

Jonas Zeitz

2

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18 March 2018

Christiane Pelzer

3

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18 March 2018

Christa Kraus

4

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18 March 2018

Andrea Peters

5

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18 March 2018

Gisela Wohlleben

6

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18 March 2018

Ingolf Berberich

7

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18 March 2018

language of work or name

English

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publication date

21 March 2006

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18 March 2018

Journal of Biological Chemistry

published in

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18 March 2018

volume

281

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18 March 2018

issue

19

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18 March 2018

page(s)

13663-13671

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18 March 2018

Apoptosis in the pathogenesis and treatment of disease

cites work

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Mitochondria and apoptosis

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Cytochrome c in the apoptotic and antioxidant cascades

21 January 2018

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The pathophysiology of mitochondrial cell death

21 January 2018

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The Bcl2 family: regulators of the cellular life-or-death switch

21 January 2018

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Keeping killers on a tight leash: transcriptional and post-translational control of the pro-apoptotic activity of BH3-only proteins

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M600266200

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M600266200

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

21 January 2018

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The role of BH3-only proteins in the immune system

21 January 2018

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Deadly encounter: ubiquitin meets apoptosis

21 January 2018

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Phosphorylation of Bim-EL by Erk1/2 on serine 69 promotes its degradation via the proteasome pathway and regulates its proapoptotic function.

21 January 2018

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Regulation of osteoclast apoptosis by ubiquitylation of proapoptotic BH3-only Bcl-2 family member Bim.

21 January 2018

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Chlamydia inhibit host cell apoptosis by degradation of proapoptotic BH3-only proteins

21 January 2018

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EBV infection of human B lymphocytes leads to down-regulation of Bim expression: relationship to resistance to apoptosis.

21 January 2018

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Posttranslational modification of Bcl-2 facilitates its proteasome-dependent degradation: molecular characterization of the involved signaling pathway.

21 January 2018

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Mule/ARF-BP1, a BH3-only E3 ubiquitin ligase, catalyzes the polyubiquitination of Mcl-1 and regulates apoptosis

21 January 2018

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The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function

21 January 2018

1 reference

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Transient expression of the Bcl-2 family member, A1-a, results in nuclear localization and resistance to staurosporine-induced apoptosis

21 January 2018

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A1 functions at the mitochondria to delay endothelial apoptosis in response to tumor necrosis factor

21 January 2018

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Characterization of the signal that directs Bcl-x(L), but not Bcl-2, to the mitochondrial outer membrane

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M600266200

Blimp-1 overcomes the block in IgM secretion in lipopolysaccharide/anti-mu F(ab')2-co-stimulated B lymphocytes.

21 January 2018

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A transient three-plasmid expression system for the production of high titer retroviral vectors

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M600266200

Bcl-2 family members: integrators of survival and death signals in physiology and pathology [corrected].

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M600266200

Discovery of the ubiquitin proteasome system and its involvement in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Mechanisms underlying ubiquitination

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

C-terminal region of Bfl-1 induces cell death that accompanies caspase activation when fused with GFP.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Proteasomes: protein degradation machines of the cell

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Constitutive proteasome-mediated turnover of Bfl-1/A1 and its processing in response to TNF receptor activation in FL5.12 pro-B cells convert it into a prodeath factor.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Context of multiubiquitin chain attachment influences the rate of Sic1 degradation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

A novel human WD protein, h-beta TrCp, that interacts with HIV-1 Vpu connects CD4 to the ER degradation pathway through an F-box motif

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

The SCFbeta-TRCP-ubiquitin ligase complex associates specifically with phosphorylated destruction motifs in IkappaBalpha and beta-catenin and stimulates IkappaBalpha ubiquitination in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Mapping of the inducible IkappaB phosphorylation sites that signal its ubiquitination and degradation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Puma(*)Mcl-1 interaction is not sufficient to prevent rapid degradation of Mcl-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600266200

21 January 2018

Identifiers

DOI

10.1074/JBC.M600266200

1 reference

18 March 2018

1 reference

18 March 2018