(Q51808507)

12 April 2018

title

Hypoxia-mediated degradation of Na,K-ATPase via mitochondrial reactive oxygen species and the ubiquitin-conjugating system. (English)

1 reference

12 April 2018

main subject

hypoxia

0 references

reactive oxygen species

1 reference

based on heuristic

inferred from title

author

Alejandro P. Comellas

object named as

Alejandro P Comellas

1

0 references

Aaron Ciechanover

9

object named as

Aaron Ciechanover

1 reference

12 April 2018

Laura A Dada

2

1 reference

12 April 2018

Emilia Lecuona

3

1 reference

12 April 2018

Liuska M Pesce

4

1 reference

12 April 2018

Navdeep S Chandel

5

1 reference

12 April 2018

Nancy Quesada

6

1 reference

12 April 2018

G R Scott Budinger

7

1 reference

12 April 2018

Ger J Strous

8

1 reference

12 April 2018

Jacob I Sznajder

10

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12 April 2018

publication date

13 April 2006

1 reference

12 April 2018

published in

Circulation Research

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12 April 2018

volume

98

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12 April 2018

issue

10

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12 April 2018

page(s)

1314-1322

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12 April 2018

Hypoxia reduces alveolar epithelial sodium and fluid transport in rats: reversal by beta-adrenergic agonist treatment

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11 October 2022

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Hormonal regulation of the Na(+)-K(+)-ATPase: mechanisms underlying rapid and sustained changes in pump activity.

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11 October 2022

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11 October 2022

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Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cells

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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Tyrosine 537 within the Na+,K+-ATPase alpha-subunit is essential for AP-2 binding and clathrin-dependent endocytosis

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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11 October 2022

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Receptor-mediated inhibition of renal Na(+)-K(+)-ATPase is associated with endocytosis of its alpha- and beta-subunits

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11 October 2022

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11 October 2022

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1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

Physiological and pathological responses to hypoxia

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11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

Molecular dissection of autophagy: two ubiquitin-like systems

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11 October 2022

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Reactive oxygen species, cell signaling, and cell injury

1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

Growth hormone-induced signal tranduction depends on an intact ubiquitin system

1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

Redox regulatory mechanisms of cellular signal transduction

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11 October 2022

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Reactive oxygen species generated at mitochondrial complex III stabilize hypoxia-inducible factor-1alpha during hypoxia: a mechanism of O2 sensing

1 reference

11 October 2022

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Salmeterol for the prevention of high-altitude pulmonary edema.

1 reference

11 October 2022

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Gettin' down with ubiquitin: turning off cell-surface receptors, transporters and channels

1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

beta-agonists regulate Na,K-ATPase via novel MAPK/ERK and rapamycin-sensitive pathways

1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1161/01.RES.0000222418.99976.1D

Identifiers

DOI

10.1161/01.RES.0000222418.99976.1D

1 reference

12 April 2018

PubMed ID

16614303

1 reference

12 April 2018