(Q53232752)

12 May 2018

title

Does dysregulation of the Notch and wingless/Wnt pathways underlie the pathogenesis of Alzheimer's disease? (English)

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12 May 2018

author

Richard Killick

3

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12 May 2018

author name string

Anderton BH

1

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12 May 2018

Dayanandan R

2

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12 May 2018

Lovestone S

4

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12 May 2018

publication date

1 February 2000

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12 May 2018

Trends in Molecular Medicine

published in

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12 May 2018

volume

6

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12 May 2018

issue

2

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12 May 2018

page(s)

54-59

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12 May 2018

Molecular pathogenesis of sporadic and familial forms of Alzheimer's disease.

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7 January 2021

Notch signaling: cell fate control and signal integration in development

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7 January 2021

A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain

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Proteolytic release and nuclear translocation of Notch-1 are induced by presenilin-1 and impaired by pathogenic presenilin-1 mutations

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A loss of function mutation of presenilin-2 interferes with amyloid beta-peptide production and notch signaling

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Presenilin 1 is required for Notch1 and DII1 expression in the paraxial mesoderm

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7 January 2021

Interaction between Wingless and Notch signaling pathways mediated by dishevelled.

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Presenilins interact with armadillo proteins including neural-specific plakophilin-related protein and beta-catenin

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Presenilin 1 interaction in the brain with a novel member of the Armadillo family

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Destabilization of beta-catenin by mutations in presenilin-1 potentiates neuronal apoptosis

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Direct association of presenilin-1 with beta-catenin

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Mutant presenilin-1 induces apoptosis and downregulates Akt/PKB.

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7 January 2021

The presenilin 1 protein is a component of a high molecular weight intracellular complex that contains beta-catenin

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7 January 2021

Evidence for a physical interaction between presenilin and Notch

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7 January 2021

Presenilin mutations associated with Alzheimer disease cause defective intracellular trafficking of beta-catenin, a component of the presenilin protein complex

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Overexpression in neurons of human presenilin-1 or a presenilin-1 familial Alzheimer disease mutant does not enhance apoptosis.

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Identifiers

DOI

1 reference

12 May 2018

1 reference

12 May 2018