(Q54379479)

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Phosphatidylserine-containing liposomes suppress inflammatory bone loss by ameliorating the cytokine imbalance provoked by infiltrated macrophages.

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Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

Phosphatidylserine-containing liposomes suppress inflammatory bone loss by ameliorating the cytokine imbalance provoked by infiltrated macrophages. (English)

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

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author name string

Hong Mei Ma

1

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

Zhou Wu

2

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

Hiroshi Nakanishi

3

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

publication date

4 April 2011

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

Laboratory Investigation

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

91

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

6

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

921-931

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

1 reference

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Resolution of inflammation: the beginning programs the end

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Resolution of inflammation: state of the art, definitions and terms

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A receptor for phosphatidylserine-specific clearance of apoptotic cells

1 reference

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Phosphatidylserine (PS) induces PS receptor-mediated macropinocytosis and promotes clearance of apoptotic cells

1 reference

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The induction of tolerance by dendritic cells that have captured apoptotic cells

1 reference

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Phosphatidylserine regulates the maturation of human dendritic cells.

1 reference

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Involvement of ERK, a MAP kinase, in the production of TGF-beta by macrophages treated with liposomes composed of phosphatidylserine

1 reference

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Involvement of COX-1 and up-regulated prostaglandin E synthases in phosphatidylserine liposome-induced prostaglandin E2 production by microglia

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Interaction between phosphatidylserine and the phosphatidylserine receptor inhibits immune responses in vivo

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Apoptotic mimicry: phosphatidylserine liposomes reduce inflammation through activation of peroxisome proliferator-activated receptors (PPARs) in vivo

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Phosphatidylserine-containing liposomes inhibit the differentiation of osteoclasts and trabecular bone loss

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Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand

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Critical roles for interleukin 1 and tumor necrosis factor alpha in antibody-induced arthritis

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Molecular and cellular basis of rheumatoid joint destruction

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IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis

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Gene therapy for rheumatoid arthritis. Lessons from animal models, including studies on interleukin-4, interleukin-10, and interleukin-1 receptor antagonist as potential disease modulators

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Interleukin-10 inhibits gram-negative-microbe-specific human receptor activator of NF-kappaB ligand-positive CD4+-Th1-cell-associated alveolar bone loss in vivo

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Possible involvement of MIP-1alpha in the recruitment of osteoclast progenitors to the distal tibia in rats with adjuvant-induced arthritis

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Leptomeningeal cells activate microglia and astrocytes to induce IL-10 production by releasing pro-inflammatory cytokines during systemic inflammation.

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Immunopathogenesis and immunotherapy in rheumatoid arthritis: an area in transition.

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Mechanisms of disease: the molecular and cellular basis of joint destruction in rheumatoid arthritis

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Cathepsin K-dependent toll-like receptor 9 signaling revealed in experimental arthritis.

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Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells

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Alteration of IL-17 related protein expressions in experimental autoimmune myocarditis and inhibition of IL-17 by IL-10-Ig fusion gene transfer

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RANK is essential for osteoclast and lymph node development

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RANK is the intrinsic hematopoietic cell surface receptor that controls osteoclastogenesis and regulation of bone mass and calcium metabolism

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Modulation of osteoclast differentiation and function by the new members of the tumor necrosis factor receptor and ligand families

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IL-23 induces receptor activator of NF-kappaB ligand expression on CD4+ T cells and promotes osteoclastogenesis in an autoimmune arthritis model

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A protein kinase involved in the regulation of inflammatory cytokine biosynthesis

1 reference

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21 January 2018

Signalling, inflammation and arthritis: NF-kappaB and its relevance to arthritis and inflammation.

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21 January 2018

Involvement of p38 MAP Kinase in the inhibitory effects of phosphatidylserine liposomes on nitric oxide production from macrophages stimulated with LPS.

1 reference

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21 January 2018

Phytoglycoprotein inhibits interleukin-1beta and interleukin-6 via p38 mitogen-activated protein kinase in lipopolysaccharide-stimulated RAW 264.7 cells

1 reference

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Reciprocal CD40 signals through p38MAPK and ERK-1/2 induce counteracting immune responses

1 reference

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ERK activation following macrophage FcgammaR ligation leads to chromatin modifications at the IL-10 locus

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The kinase p38 alpha serves cell type-specific inflammatory functions in skin injury and coordinates pro- and anti-inflammatory gene expression

1 reference

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Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation

1 reference

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7 January 2021

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Regulation of phosphatidylserine exposure and phagocytosis of apoptotic T lymphocytes

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2011.54

7 January 2021

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The central role of phosphatidylserine in the phagocytosis of apoptotic thymocytes

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7 January 2021

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Lipopolysaccharide induces rapid production of IL-10 by monocytes in the presence of apoptotic neutrophils

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7 January 2021

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Artificial phosphatidylserine liposome mimics apoptotic cells in inhibiting maturation and immunostimulatory function of murine myeloid dendritic cells in response to 1-chloro-2,4-dinitrobenze in vitro

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The combination of tumor necrosis factor alpha blockade with interleukin-1 and interleukin-17 blockade is more effective for controlling synovial inflammation and bone resorption in an ex vivo model

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Magnetic resonance imaging and spectroscopy of the periarticular inflammatory soft-tissue changes in experimental arthritis of the rat

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Involvement of sensory nerves and immune cells in osteophyte formation in the ankle joint of adjuvant arthritic rats

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Synovial tissue in rheumatoid arthritis is a source of osteoclast differentiation factor

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Endotoxin signal transduction in macrophages.

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Identifiers

10.1038/LABINVEST.2011.54

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 May 2018

http://europepmc.org/abstract/MED/21464820

ResearchGate publication ID

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