(Q54508747)

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Human umbilical cord blood mononuclear cells activate the survival protein Akt in cardiac myocytes and endothelial cells that limits apoptosis and necrosis during hypoxia.

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

Human umbilical cord blood mononuclear cells activate the survival protein Akt in cardiac myocytes and endothelial cells that limits apoptosis and necrosis during hypoxia. (English)

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

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Paul R. Sanberg

5

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Paul Sanberg

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

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Robert J Henning

1

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

Steve Dennis

2

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

Darrell Sawmiller

3

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

Lorynn Hunter

4

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

Leslie Miller

6

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PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

publication date

28 February 2012

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

Translational Research

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

159

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

6

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

497-506

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

The global burden of disease, 1990–2020

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Heart disease and stroke statistics--2011 update: a report from the American Heart Association

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Human umbilical cord blood mononuclear cells for the treatment of acute myocardial infarction

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Human umbilical cord blood progenitor cells are attracted to infarcted myocardium and significantly reduce myocardial infarction size

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Human cord blood mononuclear cells decrease cytokines and inflammatory cells in acute myocardial infarction

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Hematopoietic stem/progenitor cells, generation of induced pluripotent stem cells, and isolation of endothelial progenitors from 21- to 23.5-year cryopreserved cord blood

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CD34 stem/progenitor cells purified from cryopreserved normal cord blood can be transduced with high efficiency by a retroviral vector and expanded ex vivo with stable integration and expression of Fanconi anemia complementation C gene

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Bone marrow cells regenerate infarcted myocardium

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Proliferation, differentiation, and cytokine secretion of human umbilical cord blood-derived mononuclear cells in vitro.

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Neuroprotection by cord blood neural progenitors involves antioxidants, neurotrophic and angiogenic factors

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Numerous growth factors, cytokines, and chemokines are secreted by human CD34+ cells, myeloblasts, erythroblasts, and megakaryoblasts and regulate normal hematopoiesis in an autocrine/paracrine manner

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7 January 2021

Myocardial AKT: the omnipresent nexus

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7 January 2021

Akt activation preserves cardiac function and prevents injury after transient cardiac ischemia in vivo

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7 January 2021

Akt2: a critical regulator of cardiomyocyte survival and metabolism.

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H9c2 cell line is a valuable in vitro model to study the drug metabolizing enzymes in the heart

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Morphological, biochemical, and electrophysiological characterization of a clonal cell (H9c2) line from rat heart

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The Akt activation inhibitor TCN-P inhibits Akt phosphorylation by binding to the PH domain of Akt and blocking its recruitment to the plasma membrane

1 reference

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7 January 2021

A small molecule inhibits Akt through direct binding to Akt and preventing Akt membrane translocation

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Akt/protein kinase B signaling inhibitor-2, a selective small molecule inhibitor of Akt signaling with antitumor activity in cancer cells overexpressing Akt.

1 reference

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7 January 2021

Human umbilical cord blood cells protect oligodendrocytes from brain ischemia through Akt signal transduction.

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IGF-I differentially regulates Bcl-xL and Bax and confers myocardial protection in the rat heart

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Overexpression of insulin-like growth factor-1 in mice protects from myocyte death after infarction, attenuating ventricular dilation, wall stress, and cardiac hypertrophy

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Hepatocyte growth factor prevents ventricular remodeling and dysfunction in mice via Akt pathway and angiogenesis

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Cardioprotective effect of interleukin-10 in murine myocardial ischemia-reperfusion

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Cardioprotective actions of endogenous IL-10 are independent of iNOS

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Local myocardial insulin-like growth factor 1 (IGF-1) delivery with biotinylated peptide nanofibers improves cell therapy for myocardial infarction.

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Coexpression of VEGF and angiopoietin-1 promotes angiogenesis and cardiomyocyte proliferation reduces apoptosis in porcine myocardial infarction (MI) heart

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Akt promotes survival of cardiomyocytes in vitro and protects against ischemia-reperfusion injury in mouse heart.

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Akt in ischemia and reperfusion

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation

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Apoptosis of endothelial cells precedes myocyte cell apoptosis in ischemia/reperfusion injury

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Myocardial ischemia and reperfusion injury

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Real-time imaging of apoptotic cell-membrane changes at the single-cell level in the beating murine heart

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Early features of apoptosis detected by four different flow cytometry assays

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Fundamental aspects of the interaction of propidium diiodide with nuclei acids studied in a model system of polyacrylamide films

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Distribution of hematopoietic stem cells in the bone marrow according to regional hypoxia

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Identifiers

10.1016/J.TRSL.2012.02.004

1 reference

Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/22633101

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