(Q56889933)

inferred from title

author

Marcel Philipp Trefny

Marcel P Trefny

3

0 references

Daniela S Thommen

Daniela S Thommen

4

0 references

Frank Stenner

Frank Stenner

18

0 references

Stephan von Gunten

Stephan von Gunten

6

0 references

Alexandar Tzankov

Alexandar Tzankov

Viola Heinzelmann-Schwarz

7

0 references

10

Viola Heinzelmann-Schwarz

0 references

Christopher Amos

15

Christopher I Amos

0 references

Heinz-Josef Lenz

13

Heinz-Josef Lenz

0 references

author name string

Michal A Stanczak

1

0 references

Shoib S Siddiqui

2

0 references

Kayluz Frias Boligan

5

0 references

Lothar Tietze

8

0 references

Didier Lardinois

9

0 references

Michael von Bergwelt-Baidon

11

0 references

Wu Zhang

12

0 references

Younghun Han

14

0 references

Mohammedyaseen Syedbasha

16

0 references

Adrian Egli

17

0 references

Daniel E Speiser

19

0 references

Ajit Varki

20

0 references

Alfred Zippelius

21

0 references

Heinz Läubli

Journal of Clinical Investigation

22

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language of work or name

English

0 references

publication date

21 August 2018

0 references

published in

0 references

volume

128

0 references

issue

11

0 references

page(s)

4912-4923

0 references

describes a project that uses

ImageJ

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC6205408/fullTextXML

inferred from PubMed Central ID database lookup

cites work

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Loss of Siglec expression on T lymphocytes during human evolution

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Siglecs and their roles in the immune system

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Toll-like receptors and immune regulation: implications for cancer therapy

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Molecular mimicry of host sialylated glycans allows a bacterial pathogen to engage neutrophil Siglec-9 and dampen the innate immune response

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Pathogen recognition and inflammatory signaling in innate immune defenses

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Group B Streptococcus suppression of phagocyte functions by protein-mediated engagement of human Siglec-5.

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Sialylation of 3-methylcholanthrene-induced fibrosarcoma determines antitumor immune responses during immunoediting

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Sterile inflammation: sensing and reacting to damage

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Since there are PAMPs and DAMPs, there must be SAMPs? Glycan “self-associated molecular patterns” dampen innate immunity, but pathogens can mimic them

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Regulatory circuits mediated by lectin-glycan interactions in autoimmunity and cancer

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Cell-intrinsic mechanism involving Siglec-5 associated with divergent outcomes of HIV-1 infection in human and chimpanzee CD4 T cells

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T cell regulation mediated by interaction of soluble CD52 with the inhibitory receptor Siglec-10.

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Glycocalyx engineering reveals a Siglec-based mechanism for NK cell immunoevasion

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Rapid evolution of binding specificities and expression patterns of inhibitory CD33-related Siglecs in primates

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Group B Streptococcus engages an inhibitory Siglec through sialic acid mimicry to blunt innate immune and inflammatory responses in vivo

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Inhibitory Receptor Expression Depends More Dominantly on Differentiation and Activation than "Exhaustion" of Human CD8 T Cells

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Interactions between Siglec-7/9 receptors and ligands influence NK cell-dependent tumor immunosurveillance

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PD-1 identifies the patient-specific CD8⁺ tumor-reactive repertoire infiltrating human tumors

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In vitro characterization of the anti-PD-1 antibody nivolumab, BMS-936558, and in vivo toxicology in non-human primates

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Rare variants of large effect in BRCA2 and CHEK2 affect risk of lung cancer

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The interplay between Siglecs and sialylated pathogens

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CD22 and Siglec-G regulate inhibition of B-cell signaling by sialic acid ligand binding and control B-cell tolerance

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Engagement of myelomonocytic Siglecs by tumor-associated ligands modulates the innate immune response to cancer

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Evolving synergistic combinations of targeted immunotherapies to combat cancer.

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Molecular and cellular insights into T cell exhaustion

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Progression of Lung Cancer Is Associated with Increased Dysfunction of T Cells Defined by Coexpression of Multiple Inhibitory Receptors

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Sialic acids in cancer biology and immunity.

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Self-regulation and cross-regulation of pattern-recognition receptor signalling in health and disease

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Tumor sialylation impedes T cell mediated anti-tumor responses while promoting tumor associated-regulatory T cells

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Structural basis for sulfation-dependent self-glycan recognition by the human immune-inhibitory receptor Siglec-8

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Regulatory circuits of T cell function in cancer

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The mucin MUC1 modulates the tumor immunological microenvironment through engagement of the lectin Siglec-9.

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Influence of SIGLEC9 polymorphisms on COPD phenotypes including exacerbation frequency

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Paired Siglec receptors generate opposite inflammatory responses to a human-specific pathogen.

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Elements of cancer immunity and the cancer-immune set point

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Siglec-8 and Siglec-9 binding specificities and endogenous airway ligand distributions and properties

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PD-1 expression by tumour-associated macrophages inhibits phagocytosis and tumour immunity.

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Chromatin states define tumour-specific T cell dysfunction and reprogramming.

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Increased expression of programmed cell death protein 1 on NK cells inhibits NK-cell-mediated anti-tumor function and indicates poor prognosis in digestive cancers

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Siglec-G represses DAMP-mediated effects on T cells.

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The tumour glyco-code as a novel immune checkpoint for immunotherapy.

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