(Q56898738)

15

object named as

Ignacio Melero

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author name string

Natalia Suarez

1

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Carlos Alfaro

2

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Juan Dubrot

3

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Asis Palazon

4

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Elixabet Bolaños

5

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Lorena Erro

6

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Sandra Hervas-Stubbs

7

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Ivan Martinez-Forero

8

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Aizea Morales-Kastresana

9

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Salvador Martin-Algarra

10

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Bruno Sangro

11

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Fernando Lecanda

12

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Jose L Perez-Gracia

13

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Alvaro Gonzalez

International Journal of Cancer

14

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language of work or name

English

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publication date

15 July 2011

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published in

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volume

129

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issue

2

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page(s)

374-86

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cites work

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CD28 and CTLA-4 coreceptor expression and signal transduction

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CTLA-4 engagement inhibits IL-2 accumulation and cell cycle progression upon activation of resting T cells

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Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4

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Lymphoproliferative disorders with early lethality in mice deficient in Ctla-4

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CD4+ regulatory T cells require CTLA-4 for the maintenance of systemic tolerance

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Control of regulatory T cell lineage commitment and maintenance

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Mechanisms of foxp3+ T regulatory cell-mediated suppression

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Analysis of the cellular mechanism of antitumor responses and autoimmunity in patients treated with CTLA-4 blockade

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Cytotoxic T lymphocyte-associated molecule-4, a high-avidity receptor for CD80 and CD86, contains an intracellular localization motif in its cytoplasmic tail

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B7-1 and B7-2 selectively recruit CTLA-4 and CD28 to the immunological synapse

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CTLA-4 trafficking and surface expression

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The B7 family and cancer therapy: costimulation and coinhibition

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The interaction properties of costimulatory molecules revisited.

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The CD28 and CTLA-4 receptors associate with the serine/threonine phosphatase PP2A.

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Molecular basis of T cell inactivation by CTLA-4

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Blockade of CTLA-4 on both effector and regulatory T cell compartments contributes to the antitumor activity of anti-CTLA-4 antibodies

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CTLA-4 control over Foxp3+ regulatory T cell function

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Enhancement of antitumor immunity by CTLA-4 blockade

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Cell intrinsic mechanisms of T-cell inhibition and application to cancer therapy

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Combination immunotherapy of B16 melanoma using anti-cytotoxic T lymphocyte-associated antigen 4 (CTLA-4) and granulocyte/macrophage colony-stimulating factor (GM-CSF)-producing vaccines induces rejection of subcutaneous and metastatic tumors accomp

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Immunologic and clinical effects of antibody blockade of cytotoxic T lymphocyte-associated antigen 4 in previously vaccinated cancer patients

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Improved Survival with Ipilimumab in Patients with Metastatic Melanoma

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Anti-CTLA-4 therapy results in higher CD4+ICOShi T cell frequency and IFN-gamma levels in both nonmalignant and malignant prostate tissues

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Synergism of cytotoxic T lymphocyte-associated antigen 4 blockade and depletion of CD25(+) regulatory T cells in antitumor therapy reveals alternative pathways for suppression of autoreactive cytotoxic T lymphocyte responses

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Combination of tumor site-located CTL-associated antigen-4 blockade and systemic regulatory T-cell depletion induces tumor-destructive immune responses.

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Influence of bevacizumab, sunitinib and sorafenib as single agents or in combination on the inhibitory effects of VEGF on human dendritic cell differentiation from monocytes

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Phase I/II trial of tremelimumab in patients with metastatic melanoma

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Generation of Epstein-Barr Virus (EBV)-immortalized B cell lines

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The combined actions of NK and T lymphocytes are necessary to reject an EGFP+ mesenchymal tumor through mechanisms dependent on NKG2D and IFN gamma.

21 January 2018

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Low surface expression of B7-1 (CD80) is an immunoescape mechanism of colon carcinoma

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Tumor-reactive CD4(+) T cells develop cytotoxic activity and eradicate large established melanoma after transfer into lymphopenic hosts

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Long-term outcome of EBV-specific T-cell infusions to prevent or treat EBV-related lymphoproliferative disease in transplant recipients

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Treatment of nasopharyngeal carcinoma with Epstein-Barr virus--specific T lymphocytes

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Reversal of the TCR stop signal by CTLA-4

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Altering regulatory T cell function in cancer immunotherapy: a novel means to boost the efficacy of cancer vaccines

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CD4 cells can be more efficient at tumor rejection than CD8 cells

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CTLA-4 blockade enhances polyfunctional NY-ESO-1 specific T cell responses in metastatic melanoma patients with clinical benefit

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Anti-CTLA4 antibody-induced lupus nephritis

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CTLA-4 on alloreactive CD4 T cells interacts with recipient CD80/86 to promote tolerance

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