(Q56930964)

English

Life can be stressful without ATR

No description defined

In more languages

edit

Statements

instance of

scholarly article

0 references

title

Life can be stressful without ATR (English)

0 references

Mark O'Driscoll

1

0 references

language of work or name

English

0 references

publication date

August 2009

0 references

published in

Nature Genetics

0 references

volume

41

0 references

issue

8

0 references

page(s)

866-868

0 references

exact match

https://scigraph.springernature.com/pub.10.1038/ng0809-866

0 references

cites work

Targeted disruption of the cell-cycle checkpoint gene ATR leads to early embryonic lethality in mice

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

A splicing mutation affecting expression of ataxia-telangiectasia and Rad3-related protein (ATR) results in Seckel syndrome

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

Mutations in pericentrin cause Seckel syndrome with defective ATR-dependent DNA damage signaling

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

Seckel syndrome exhibits cellular features demonstrating defects in the ATR-signalling pathway.

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

The role of the DNA damage response pathways in brain development and microcephaly: insight from human disorders

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

Deletion of the developmentally essential gene ATR in adult mice leads to age-related phenotypes and stem cell loss

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

Persistent transcription-blocking DNA lesions trigger somatic growth attenuation associated with longevity

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

ATR functions as a gene dosage-dependent tumor suppressor on a mismatch repair-deficient background

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

Chk1 is haploinsufficient for multiple functions critical to tumor suppression

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

A viable allele of Mcm4 causes chromosome instability and mammary adenocarcinomas in mice.

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

Mutation in Rpa1 results in defective DNA double-strand break repair, chromosomal instability and cancer in mice

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2FNG0809-866

retrieved

21 January 2018

ATR disruption leads to chromosomal fragmentation and early embryonic lethality

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/19639025

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A mouse model of ATR-Seckel shows embryonic replicative stress and accelerated aging

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/19639025

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

DOI

10.1038/NG0809-866

0 references

PubMed publication ID

19639025

0 references

(Q56930964)

Life can be stressful without ATR

Statements

Identifiers

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)