(Q57220366)

Marie-Thérèse Dimanche-Boitrel

7

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Olivier Micheau

Olivier Micheau

2

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Eric Solary

Eric Solary

6

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Jürg Tschopp

5

Jurg Tschopp

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author name string

Sandrine Lacour

1

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Arlette Hammann

3

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Véronique Drouineaud

https://scigraph.springernature.com/pub.10.1038/sj.onc.1206127

4

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language of work or name

English

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publication date

March 2003

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published in

Oncogene

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volume

22

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issue

12

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page(s)

1807-1816

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exact match

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cites work

Safety and antitumor activity of recombinant soluble Apo2 ligand

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Modulation of life and death by the TNF receptor superfamily

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TRAIL receptor-2 signals apoptosis through FADD and caspase-8

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Evaluation of multiple bio-pathological factors in colorectal adenocarcinomas: independent prognostic role of p53 and bcl-2.

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Clinical pharmacology of recombinant human tumor necrosis factor in patients with advanced cancer

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Death receptor 5, a new member of the TNFR family, and DR4 induce FADD-dependent apoptosis and activate the NF-kappaB pathway

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TRAIL-induced apoptosis requires Bax-dependent mitochondrial release of Smac/DIABLO.

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Upregulation of CASP genes in human tumor cells undergoing etoposide-induced apoptosis.

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Therapeutic modulation of death receptor- or drug-mediated apoptosis

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Increased expression of death receptors 4 and 5 synergizes the apoptosis response to combined treatment with etoposide and TRAIL

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Mitochondria and apoptosis.

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An essential role for membrane rafts in the initiation of Fas/CD95-triggered cell death in mouse thymocytes

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Roles of lipid rafts in membrane transport

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An inducible pathway for degradation of FLIP protein sensitizes tumor cells to TRAIL-induced apoptosis

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Death receptor recruitment of endogenous caspase-10 and apoptosis initiation in the absence of caspase-8

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Mitochondrial control of cell death

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Membrane oligomerization and cleavage activates the caspase-8 (FLICE/MACHalpha1) death signal

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STAT-1-independent upregulation of FADD and procaspase-3 and -8 in cancer cells treated with cytotoxic drugs.

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NF-kappaB signals induce the expression of c-FLIP.

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Fas ligand-independent, FADD-mediated activation of the Fas death pathway by anticancer drugs.

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CCNU-dependent potentiation of TRAIL/Apo2L-induced apoptosis in human glioma cells is p53-independent but may involve enhanced cytochrome c release

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Two CD95 (APO-1/Fas) signaling pathways

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TRAIL receptors 1 (DR4) and 2 (DR5) signal FADD-dependent apoptosis and activate NF-kappaB

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Control of TRAIL-induced apoptosis by a family of signaling and decoy receptors

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Intracellular mechanisms of TRAIL: apoptosis through mitochondrial-dependent and -independent pathways

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Overexpression of BCL2 blocks TNF-related apoptosis-inducing ligand (TRAIL)-induced apoptosis in human lung cancer cells

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Tumoricidal activity of tumor necrosis factor-related apoptosis-inducing ligand in vivo.

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Identification and characterization of a new member of the TNF family that induces apoptosis

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TRAIL causes cleavage of bid by caspase-8 and loss of mitochondrial membrane potential resulting in apoptosis in BJAB cells

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Chemotherapeutic agents augment TRAIL-induced apoptosis in human hepatocellular carcinoma cell lines

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