(Q57384599)

6

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Claudia Friesen

1

object named as

Claudia Friesen

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author name string

Annelie Lubatschofski

2

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J�rg Kotzerke

3

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Inga Buchmann

4

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Sven N. Reske

European Journal of Nuclear Medicine and Molecular Imaging

5

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publication date

1 September 2003

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published in

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volume

30

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issue

9

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page(s)

1251-1261

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exact match

https://scigraph.springernature.com/pub.10.1007/s00259-003-1216-z

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cites work

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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Drug-induced apoptosis is associated with enhanced Fas (Apo-1/CD95) ligand expression but occurs independently of Fas (Apo-1/CD95) signaling in human T-acute lymphatic leukemia cells

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HMBA induces activation of a caspase-independent cell death pathway to overcome P-glycoprotein-mediated multidrug resistance

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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Topoisomerase-I inhibitors for human malignant glioma: differential modulation of p53, p21, bax and bcl-2 expression and of CD95-mediated apoptosis by camptothecin and beta-lapachone.

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The proto-oncogene Bcl-2 and its role in regulating apoptosis

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Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

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Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

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Two CD95 (APO-1/Fas) signaling pathways

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Cleavage of human inhibitor of apoptosis protein XIAP results in fragments with distinct specificities for caspases

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Regulation of p53 in response to DNA damage

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Induction of apoptosis by cancer chemotherapy.

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Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

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Anti-cancer-prostaglandin-induced cell-cycle arrest and its modulation by an inhibitor of the ATP-dependent glutathione S-conjugate export pump (GS-X pump).

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DNA damage, p53 and anticancer therapies

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Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor

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Targeted bone marrow irradiation in the conditioning of high-risk leukaemia prior to stem cell transplantation

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Disturbances of the CD95 (APO-1/Fas) system in disorders of lymphohaematopoietic cells

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

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Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

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Anticancer drugs induce caspase-8/FLICE activation and apoptosis in the absence of CD95 receptor/ligand interaction.

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Radiolabeled anti-CD45 monoclonal antibodies target lymphohematopoietic tissue in the macaque

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Induction of CD95 ligand and apoptosis by doxorubicin is modulated by the redox state in chemosensitive- and drug-resistant tumor cells

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Detection and characterization of apoptotic peripheral blood lymphocytes in human immunodeficiency virus infection and cancer chemotherapy by a novel flow immunocytometric method.

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Chemotherapeutic drug-induced apoptosis in human leukaemic cells is independent of the Fas (APO-1/CD95) receptor/ligand system.

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The cellular response to p53: the decision between life and death

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A conserved XIAP-interaction motif in caspase-9 and Smac/DIABLO regulates caspase activity and apoptosis

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Molecular characterization of mitochondrial apoptosis-inducing factor

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Activation of apoptosis pathways in peripheral blood lymphocytes by in vivo chemotherapy

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CD95 (APO-1/Fas)-associating signalling proteins

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