(Q58198513)

7

object named as

Stanley F. Nelson

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author name string

Scherise Mitchell

1

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Asuka Ota

2

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William Foster

3

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Bin Zhang

4

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Zixing Fang

5

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Shilpa Patel

6

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Steve Horvath

STRESS signaling pathways that modulate cardiac myocyte apoptosis

8

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publication date

13 March 2006

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published in

Physiological Genomics

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volume

25

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issue

1

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page(s)

50-59

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cites work

1 reference

https://api.crossref.org/works/10.1152%2FPHYSIOLGENOMICS.00224.2005

Cardiac excitation-contraction coupling

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Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death

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Role of the stress-activated protein kinases in endothelin-induced cardiomyocyte hypertrophy

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GenMAPP, a new tool for viewing and analyzing microarray data on biological pathways

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MAP kinase kinase 6-p38 MAP kinase signaling cascade regulates cyclooxygenase-2 expression in cardiac myocytes in vitro and in vivo

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Cyclooxygenase-2 inhibitor treatment improves left ventricular function and mortality in a murine model of doxorubicin-induced heart failure

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Manipulating cardiac contractility in heart failure: data from mice and men.

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Gene expression profiling of gliomas strongly predicts survival

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Hypertrophy of the heart: a new therapeutic target?

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Cardiac hypertrophy: the good, the bad, and the ugly

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Ras reduces L-type calcium channel current in cardiac myocytes. Corrective effects of L-channels and SERCA2 on [Ca(2+)](i) regulation and cell morphology

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The Raf-MEK-ERK cascade represents a common pathway for alteration of intracellular calcium by Ras and protein kinase C in cardiac myocytes.

21 January 2018

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Identifying biological themes within lists of genes with EASE

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p38 MAP Kinase Mediates Inflammatory Cytokine Induction in Cardiomyocytes and Extracellular Matrix Remodeling in Heart

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c-Jun N-terminal kinases (JNK) antagonize cardiac growth through cross-talk with calcineurin-NFAT signaling

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Redefining the roles of p38 and JNK signaling in cardiac hypertrophy: dichotomy between cultured myocytes and animal models

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The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy

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p38 Mitogen-activated protein kinase mediates a negative inotropic effect in cardiac myocytes

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Signalling components involved in contraction-inducible substrate uptake into cardiac myocytes

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Regulation of cardiac long-chain fatty acid and glucose uptake by translocation of substrate transporters

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Cytoplasmic signaling pathways that regulate cardiac hypertrophy

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Uncoupling proteins in human heart

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Targeted activation of c-Jun N-terminal kinase in vivo induces restrictive cardiomyopathy and conduction defects

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c-Jun N-terminal kinase activation mediates downregulation of connexin43 in cardiomyocytes

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Stress-activated MAP kinases in cardiac remodeling and heart failure; new insights from transgenic studies

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TGF-beta1 and angiotensin networking in cardiac remodeling

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Peroxisome proliferator-activated receptors and cardiovascular remodeling

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Altered Na/Ca exchange activity in cardiac hypertrophy and heart failure: a new target for therapy?

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Temporally regulated and tissue-specific gene manipulations in the adult and embryonic heart using a tamoxifen-inducible Cre protein

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Peroxisome proliferator-activated receptors: lipid binding proteins controling gene expression

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Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family

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Cardiac hypertrophy induced by mitogen-activated protein kinase kinase 7, a specific activator for c-Jun NH2-terminal kinase in ventricular muscle cells

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The molecular mechanism of cardiac hypertrophy and failure

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Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart.

21 January 2018

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