(Q24298215)

positive regulation of extrinsic apoptotic signaling pathway in absence of ligand

GOA release 2020-03-11

1 reference

Receptor (TNFRSF)-interacting serine-threonine kinase 1

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author

Lieselotte Vande Walle

1

0 references

Peter Vandenabeele

8

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Tom Vanden Berghe

object named as

Tom Vanden Berghe

7

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Nele Festjens

object named as

Nele Festjens

4

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Xavier Saelens

6

object named as

Xavier Saelens

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author name string

Ellen Wirawan

2

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Mohamed Lamkanfi

3

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Jelle Verspurten

Activation of death-inducing signaling complex (DISC) by pro-apoptotic C-terminal fragment of RIP

5

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language of work or name

English

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publication date

April 2010

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published in

Cell Research

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volume

20

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issue

4

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page(s)

421-33

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cites work

1 reference

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

20 April 2017

1 reference

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

20 April 2017

1 reference

Loss of Omi mitochondrial protease activity causes the neuromuscular disorder of mnd2 mutant mice

20 April 2017

1 reference

Neuroprotective role of the Reaper-related serine protease HtrA2/Omi revealed by targeted deletion in mice

20 April 2017

1 reference

Loss of function mutations in the gene encoding Omi/HtrA2 in Parkinson's disease

20 April 2017

1 reference

Identification of Omi/HtrA2 as a mitochondrial apoptotic serine protease that disrupts inhibitor of apoptosis protein-caspase interaction

20 April 2017

1 reference

The serine protease Omi/HtrA2 regulates apoptosis by binding XIAP through a reaper-like motif

20 April 2017

1 reference

A serine protease, HtrA2, is released from the mitochondria and interacts with XIAP, inducing cell death

20 April 2017

1 reference

The serine protease Omi/HtrA2 is released from mitochondria during apoptosis. Omi interacts with caspase-inhibitor XIAP and induces enhanced caspase activity

20 April 2017

1 reference

HtrA2 promotes cell death through its serine protease activity and its ability to antagonize inhibitor of apoptosis proteins

20 April 2017

1 reference

Omi/HtrA2 catalytic cleavage of inhibitor of apoptosis (IAP) irreversibly inactivates IAPs and facilitates caspase activity in apoptosis

20 April 2017

1 reference

Omi/HtrA2 promotes cell death by binding and degrading the anti-apoptotic protein ped/pea-15

20 April 2017

1 reference

Tumor necrosis factor signaling to stress-activated protein kinase (SAPK)/Jun NH2-terminal kinase (JNK) and p38. Germinal center kinase couples TRAF2 to mitogen-activated protein kinase/ERK kinase kinase 1 and SAPK while receptor interacting [...]

20 April 2017

1 reference

EDAG regulates the proliferation and differentiation of hematopoietic cells and resists cell apoptosis through the activation of nuclear factor-kappa B

20 April 2017

1 reference

JNK suppresses apoptosis via phosphorylation of the proapoptotic Bcl-2 family protein BAD

20 April 2017

1 reference

Proteome-wide Identification of HtrA2/Omi Substrates

20 April 2017

1 reference

Identification and characterization of a novel cell cycle-regulated internal ribosome entry site

20 April 2017

1 reference

Activators and target genes of Rel/NF-kappaB transcription factors

20 April 2017

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

Activation of a pro-apoptotic amplification loop through inhibition of NF-kappaB-dependent survival signals by caspase-mediated inactivation of RIP

20 April 2017

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Rip1 mediates the Trif-dependent toll-like receptor 3- and 4-induced NF-{kappa}B activation but does not contribute to interferon regulatory factor 3 activation

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

The death domain kinase RIP has an essential role in DNA damage-induced NF-kappa B activation

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

RIP1 is an essential mediator of Toll-like receptor 3-induced NF-kappa B activation

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Apoptosis in hematopoietic cells (FL5.12) caused by interleukin-3 withdrawal: relationship to caspase activity and the loss of glutathione

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Apoptosis of hematopoietic cells induced by growth factor withdrawal is associated with caspase-9 mediated cleavage of Raf-1.

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Apaf-1 and caspase-9 accelerate apoptosis, but do not determine whether factor-deprived or drug-treated cells die

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Cell death independent of caspases: a review.

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Toxic proteins released from mitochondria in cell death

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Regulation of HAX-1 anti-apoptotic protein by Omi/HtrA2 protease during cell death

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

The role of the death-domain kinase RIP in tumour-necrosis-factor-induced activation of mitogen-activated protein kinases

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

BCL-2 in the crosshairs: tipping the balance of life and death

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Regulation of survival pathways by IL-3 and induction of apoptosis following IL-3 withdrawal

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Evidence for a role of NF-kappaB in the survival of hematopoietic cells mediated by interleukin 3 and the oncogenic TEL/platelet-derived growth factor receptor beta fusion protein

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

NF-kappaB family proteins participate in multiple steps of hematopoiesis through elimination of reactive oxygen species

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Apoptosis overrides survival signals through a caspase-mediated dominant-negative NF-kappa B loop

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Signal transduction by tumor necrosis factor and its relatives

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

p38 MAP kinases: key signalling molecules as therapeutic targets for inflammatory diseases

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021

Dual signaling of the Fas receptor: initiation of both apoptotic and necrotic cell death pathways

1 reference

https://api.crossref.org/works/10.1038%2FCR.2010.18

7 January 2021