(Q24301910)

1

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Yuefeng Wang

2

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Jeremy Post

3

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Amanda Nourse

4

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William Webb

5

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Marina Mapelli

6

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Richard W Kriwacki

Journal of Molecular Biology

9

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language of work or name

English

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publication date

17 June 2005

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published in

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volume

349

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issue

4

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page(s)

764-73

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cites work

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Intrinsically unstructured proteins: re-assessing the protein structure-function paradigm

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Calculation of protein extinction coefficients from amino acid sequence data

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Substrates for p34cdc2: in vivo veritas?

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The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

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WAF1, a potential mediator of p53 tumor suppression

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p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21

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MOLMOL: a program for display and analysis of macromolecular structures

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Cdk5 deregulation in the pathogenesis of Alzheimer's disease

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Immunohistochemical examination of the INK4 and Cip inhibitors in the rat neonatal cerebellum: cellular localization and the impact of protein calorie malnutrition.

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Probing protein structure using biochemical and biophysical methods. Proteolysis, matrix-assisted laser desorption/ionization mass spectrometry, high-performance liquid chromatography and size-exclusion chromatography of p21Waf1/Cip1/Sdi1.

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Kinetic analysis of macromolecular interactions using surface plasmon resonance biosensors

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Structure and regulation of the CDK5-p25(nck5a) complex

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Spectroscopic Determination of Tryptophan and Tyrosine in Proteins*

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Intrinsic disorder and protein function

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Probing Protein/Protein Interactions with Mass Spectrometry and Isotopic Labeling: Analysis of the p21/Cdk2 Complex

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p21 is a universal inhibitor of cyclin kinases

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Inhibition of CDK2 activity in vivo by an associated 20K regulatory subunit.

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Principles of CDK regulation

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Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration

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p27 binds cyclin-CDK complexes through a sequential mechanism involving binding-induced protein folding

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The brain-specific activator p35 allows Cdk5 to escape inhibition by p27Kip1 in neurons.

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Inhibition of cyclin-dependent kinases by p21

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The role of Cdk7 in CAK function, a retro-retrospective

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p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest

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Cloning of p57KIP2, a cyclin-dependent kinase inhibitor with unique domain structure and tissue distribution

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p57KIP2, a structurally distinct member of the p21CIP1 Cdk inhibitor family, is a candidate tumor suppressor gene

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