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A Mitochondrial Perspective of Chronic Obstructive Pulmonary Disease Pathogenesis
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scholarly article
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PubMed
review article
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Europe PubMed Central
title
A Mitochondrial Perspective of Chronic Obstructive Pulmonary Disease Pathogenesis
(English)
1 reference
stated in
PubMed
main subject
chronic obstructive pulmonary disease
0 references
pathogenesis
1 reference
based on heuristic
inferred from title
author name string
Min-Jong Kang
series ordinal
1
1 reference
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Crossref
Gerald S. Shadel
series ordinal
2
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stated in
Crossref
language of work or name
English
1 reference
stated in
PubMed
publication date
October 2016
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stated in
PubMed
published in
Tuberculosis and respiratory diseases
1 reference
stated in
PubMed
volume
79
1 reference
stated in
PubMed
issue
4
1 reference
stated in
PubMed
page(s)
207-213
1 reference
stated in
PubMed
copyright license
Creative Commons Attribution-NonCommercial 4.0 International
start time
1 January 2016
1 reference
stated in
April 2022 Public Data File from Crossref
copyright status
copyrighted
0 references
cites work
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Global Strategy for the Diagnosis, Management, and Prevention of Chronic Obstructive Pulmonary Disease
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NLRX1 protein attenuates inflammatory responses to infection by interfering with the RIG-I-MAVS and TRAF6-NF-κB signaling pathways
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The Innate Immune Receptor NLRX1 Functions as a Tumor Suppressor by Reducing Colon Tumorigenesis and Key Tumor-Promoting Signals
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Mitochondrial Regulation of Inflammasome Activation in Chronic Obstructive Pulmonary Disease
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Mitochondrial ROS signaling in organismal homeostasis
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29 September 2017
Pulmonary macrophages: key players in the innate defence of the airways.
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29 September 2017
Suppression of NLRX1 in chronic obstructive pulmonary disease
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29 September 2017
Implication of Interleukin (IL)-18 in the pathogenesis of chronic obstructive pulmonary disease (COPD).
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Accelerated ageing of the lung in COPD: new concepts.
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29 September 2017
COPD: balancing oxidants and antioxidants
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29 September 2017
Role of alveolar macrophages in chronic obstructive pulmonary disease.
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29 September 2017
Organelle-specific initiation of cell death
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29 September 2017
Mitochondria are required for antigen-specific T cell activation through reactive oxygen species signaling.
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29 September 2017
Immunopathogenesis of chronic obstructive pulmonary disease.
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29 September 2017
Mitochondria: master regulators of danger signalling
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29 September 2017
Modulating mitochondrial intracellular location as a redox signal
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29 September 2017
The mitochondrial proteins NLRX1 and TUFM form a complex that regulates type I interferon and autophagy.
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29 September 2017
IL-18 induces emphysema and airway and vascular remodeling via IFN-γ, IL-17A, and IL-13
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29 September 2017
New insights into the immunology of chronic obstructive pulmonary disease.
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29 September 2017
Mitochondria in innate immunity
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29 September 2017
Mitochondria in innate immune responses
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The aging lung and chronic obstructive pulmonary disease: similarity and difference
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PubMed Central
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29 September 2017
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29 September 2017
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29 September 2017
Cigarette smoke selectively enhances viral PAMP- and virus-induced pulmonary innate immune and remodeling responses in mice.
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29 September 2017
NLRX1 is a regulator of mitochondrial antiviral immunity
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29 September 2017
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29 September 2017
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29 September 2017
Alveolar macrophage elimination in vivo is associated with an increase in pulmonary immune response in mice
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IL-18 is induced and IL-18 receptor alpha plays a critical role in the pathogenesis of cigarette smoke-induced pulmonary emphysema and inflammation.
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PULMONARY EMPHYSEMA AND ALPHA1-ANTITRYPSIN DEFICIENCY.
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Pathologic changes in the peripheral airways of young cigarette smokers
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Correlation of lung surface area to apoptosis and proliferation in human emphysema
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12 December 2020
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Identifiers
DOI
10.4046/TRD.2016.79.4.207
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
3125465
Fatcat ID
release_jzzs5244z5cfziazf2tq6ipnqu
1 reference
stated in
Fatcat
reference URL
https://api.fatcat.wiki/v0/release/jzzs5244z5cfziazf2tq6ipnqu
retrieved
24 November 2022
based on heuristic
mapped directly with Wikidata item
OpenCitations bibliographic resource ID
3125465
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
3125465
PMCID
5077724
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
3125465
PubMed ID
27790272
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
3125465
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