(Q28278834)

1

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S Liu

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M Fliegauf

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M Lübbert

4

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M H Werner

ETO, a target of t(8;21) in acute leukemia, makes distinct contacts with multiple histone deacetylases and binds mSin3A through its oligomerization domain

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language of work or name

English

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publication date

2 March 2006

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published in

Oncogene

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volume

25

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issue

9

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page(s)

1349-57

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cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

c-Myc target gene specificity is determined by a post-DNAbinding mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Dichotomy of AML1-ETO functions: growth arrest versus block of differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Williams-Beuren syndrome critical region-5/non-T-cell activation linker: a novel target gene of AML1/ETO.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Expression of the neutrophil elastase gene during human bone marrow cell differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Transcriptional regulation of granulocyte and monocyte development

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Aberrant recruitment of the nuclear receptor corepressor-histone deacetylase complex by the acute myeloid leukemia fusion partner ETO.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

AML1/MTG8 oncogene suppression by small interfering RNAs supports myeloid differentiation of t(8;21)-positive leukemic cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Multiple regions of ETO cooperate in transcriptional repression

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

PU.1 (Spi-1) and C/EBP alpha regulate expression of the granulocyte-macrophage colony-stimulating factor receptor alpha gene.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Mutations in ELA2, encoding neutrophil elastase, define a 21-day biological clock in cyclic haematopoiesis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

21 January 2018

ETO interacting proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Reciprocal roles for CCAAT/enhancer binding protein (C/EBP) and PU.1 transcription factors in Langerhans cell commitment

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Neutrophil elastase cleaves PML-RARalpha and is important for the development of acute promyelocytic leukemia in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Neutrophil elastase is important for PML-retinoic acid receptor alpha activities in early myeloid cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The nuclear receptor co-repressor (N-CoR) utilizes repression domains I and III for interaction and co-repression with ETO.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Lymphoid Enhancer Factor-1 Links Two Hereditary Leukemia Syndromes through Core-binding Factor α Regulation of ELA2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The t(8;21) fusion protein, AML1 ETO, specifically represses the transcription of the p14(ARF) tumor suppressor in acute myeloid leukemia.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Oncogenic transcription factors in the human acute leukemias

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

A mechanism of repression by acute myeloid leukemia-1, the target of multiple chromosomal translocations in acute leukemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

ETO, a target of t(8;21) in acute leukemia, interacts with the N-CoR and mSin3 corepressors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

AML1 interconnected pathways of leukemogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Oligomerization of RAR and AML1 transcription factors as a novel mechanism of oncogenic activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

t(8;21) breakpoints on chromosome 21 in acute myeloid leukemia are clustered within a limited region of a single gene, AML1

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

PEBP2/CBF, the murine homolog of the human myeloid AML1 and PEBP2 beta/CBF beta proto-oncoproteins, regulates the murine myeloperoxidase and neutrophil elastase genes in immature myeloid cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

C/EBP, c-Myb, and PU.1 cooperate to regulate the neutrophil elastase promoter

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

AML1-ETO downregulates the granulocytic differentiation factor C/EBPalpha in t(8;21) myeloid leukemia.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The 8;21 translocation in leukemogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Multiple functional domains of AML1: PU.1 and C/EBPalpha synergize with different regions of AML1

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

ETO, fusion partner in t(8;21) acute myeloid leukemia, represses transcription by interaction with the human N-CoR/mSin3/HDAC1 complex

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The t(8;21) fusion product, AML-1-ETO, associates with C/EBP-alpha, inhibits C/EBP-alpha-dependent transcription, and blocks granulocytic differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The t(8;21) translocation converts AML1 into a constitutive transcriptional repressor

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Deletion of an AML1-ETO C-terminal NcoR/SMRT-interacting region strongly induces leukemia development

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

CCAAT enhancer-binding protein (C/EBP) and AML1 (CBF alpha2) synergistically activate the macrophage colony-stimulating factor receptor promoter

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Oligomerization of ETO is obligatory for corepressor interaction

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

E protein silencing by the leukemogenic AML1-ETO fusion protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

The myeloid master regulator transcription factor PU.1 is inactivated by AML1-ETO in t(8;21) myeloid leukemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

Mutations in the ELA2 gene encoding neutrophil elastase are present in most patients with sporadic severe congenital neutropenia but only in some patients with the familial form of the disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

inferred from DOI database lookup

7 January 2021

based on heuristic

Interaction of MyoD family proteins with enhancers of acetylcholine receptor subunit genes in vivo

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181

inferred from DOI database lookup

7 January 2021

based on heuristic

An enhancer located between the neutrophil elastase and proteinase 3 promoters is activated by Sp1 and an Ets factor

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1209181