(Q28298644)

2

Emma Guttman-Yassky

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Mayte Suárez-Fariñas

3

Mayte Suárez-Fariñas

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James G. Krueger

8

James G Krueger

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Andrea Chiricozzi

1

Andrea Chiricozzi

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Kristine E Nograles

4

Kristine E Nograles

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Irma R Cardinale

6

Irma Cardinale

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author name string

Sergio Chimenti

Journal of Investigative Dermatology

7

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language of work or name

English

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publication date

March 2011

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published in

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volume

131

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issue

3

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page(s)

677-87

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cites work

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Oncostatin M Secreted by Skin Infiltrating T Lymphocytes Is a Potent Keratinocyte Activator Involved in Skin Inflammation

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Alefacept reduces infiltrating T cells, activated dendritic cells, and inflammatory genes in psoriasis vulgaris

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Genome-wide association scan yields new insights into the immunopathogenesis of psoriasis

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Targeting tumor necrosis factor alpha in psoriasis and psoriatic arthritis

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The role of interleukin-17 in the pathogenesis of rheumatoid arthritis

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TNF Inhibition Rapidly Down-Regulates Multiple Proinflammatory Pathways in Psoriasis Plaques

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Low expression of the IL-23/Th17 pathway in atopic dermatitis compared to psoriasis

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Th17 cytokines stimulate CCL20 expression in keratinocytes in vitro and in vivo: implications for psoriasis pathogenesis

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Up-regulation of macrophage inflammatory protein-3 alpha/CCL20 and CC chemokine receptor 6 in psoriasis.

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Th17 cells at the crossroads of innate and adaptive immunity against infectious diseases at the mucosa

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A human interleukin-12/23 monoclonal antibody for the treatment of psoriasis

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Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

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Pathogenesis and therapy of psoriasis

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Psoriasis Vulgaris Lesions Contain Discrete Populations of Th1 and Th17 T Cells

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Distinct functional motifs within the IL-17 receptor regulate signal transduction and target gene expression

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Early response cytokines and innate immunity: essential roles for TNF receptor 1 and type I IL-1 receptor during Escherichia coli pneumonia in mice

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TGF-beta is necessary for induction of IL-23R and Th17 differentiation by IL-6 and IL-23

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Polymorphisms of the IL12B and IL23R genes are associated with psoriasis

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IL-17-producing CD8+ T lymphocytes from psoriasis skin plaques are cytotoxic effector cells that secrete Th17-related cytokines.

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Functional Cooperation between Interleukin-17 and Tumor Necrosis Factor-α Is Mediated by CCAAT/Enhancer-binding Protein Family Members

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Effective treatment of psoriasis with etanercept is linked to suppression of IL-17 signaling, not immediate response TNF genes

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Identifiers

DOI

10.1038/JID.2010.340

1 reference

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

1020862434

0 references

Fatcat ID

release_atx3aoxfrrhy5deuxgfnhlgc3m

0 references

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

21085185

1 reference

Consolidated OpenCitations Corpus – April 2017