(Q28564150)

2

1 reference

31 January 2017

Ana Kilić

3

1 reference

31 January 2017

Asad Zeidan

4

1 reference

31 January 2017

Angel Choi

5

1 reference

31 January 2017

Morris Karmazyn

Journal of Molecular and Cellular Cardiology

6

1 reference

31 January 2017

1 June 2009

1 reference

31 January 2017

1 reference

31 January 2017

46

1 reference

31 January 2017

6

1 reference

31 January 2017

998–1007

1 reference

31 January 2017

Sodium-hydrogen exchanger, cardiac overload, and myocardial hypertrophy

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

The role of NHE-1 in myocardial hypertrophy and remodelling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

NHE-1 inhibition: from protection during acute ischaemia/reperfusion to prevention/reversal of myocardial remodelling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Inhibition of the Na+/H+ exchanger attenuates the deterioration of ventricular function during pacing-induced heart failure in rabbits

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Inhibition and reversal of myocardial infarction-induced hypertrophy and heart failure by NHE-1 inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Regression of hypertensive myocardial fibrosis by Na(+)/H(+) exchange inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Na+/H+ exchange inhibition attenuates left ventricular remodeling and preserves systolic function in pressure-overloaded hearts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

NHE-1 inhibition improves impaired mitochondrial permeability transition and respiratory function during postinfarction remodelling in the rat.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

The mitochondrial permeability transition from in vitro artifact to disease target

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Antihypertrophic effect of Na+/H+ exchanger isoform 1 inhibition is mediated by reduced mitogen-activated protein kinase activation secondary to improved mitochondrial integrity and decreased generation of mitochondrial-derived reactive oxygen speci

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

7 January 2021

The renaissance of GSK3

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

The multifaceted roles of glycogen synthase kinase 3beta in cellular signaling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Glycogen synthase kinase-3beta is a negative regulator of cardiomyocyte hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Glycogen synthase kinase-3beta: a novel regulator of cardiac hypertrophy and development.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Inhibition of GSK-3beta as a target for cardioprotection: the importance of timing, location, duration and degree of inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Modulation of the mitochondrial permeability transition pore complex in GSK-3beta-mediated myocardial protection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

AMP-activated protein kinase (AMPK) activating agents cause dephosphorylation of Akt and glycogen synthase kinase-3.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

AICAR positively regulate glycogen synthase activity and LDL receptor expression through Raf-1/MEK/p42/44MAPK/p90RSK/GSK-3 signaling cascade

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

AMP-activated protein kinase in the heart: role during health and disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

AMPK alterations in cardiac physiology and pathology: enemy or ally?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Na(+)/H(+) exchange inhibition attenuates hypertrophy and heart failure in 1-wk postinfarction rat myocardium

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Adiponectin-mediated modulation of hypertrophic signals in the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Insulin antagonizes ischemia-induced Thr172 phosphorylation of AMP-activated protein kinase alpha-subunits in heart via hierarchical phosphorylation of Ser485/491.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

The AMP-activated protein kinase pathway--new players upstream and downstream.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Increased adenosine monophosphate-activated protein kinase activity in rat hearts with pressure-overload hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Activation of AMP-activated protein kinase inhibits protein synthesis associated with hypertrophy in the cardiac myocyte

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Adiponectin protects against myocardial ischemia-reperfusion injury through AMPK- and COX-2-dependent mechanisms

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

AICAR inhibits the Na+/H+ exchanger in rat hearts--possible contribution to cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

5-aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside reduces glucose uptake via the inhibition of Na+/H+ exchanger 1 in isolated rat ventricular cardiomyocytes.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Enhanced activity of the myocardial Na+/H+ exchanger NHE-1 contributes to cardiac remodeling in atrial natriuretic peptide receptor-deficient mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Bradykinin prevents reperfusion injury by targeting mitochondrial permeability transition pore through glycogen synthase kinase 3beta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Mitochondrial VDAC can be phosphorylated by cyclic AMP-dependent protein kinase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

Activation of glycogen synthase kinase 3beta disrupts the binding of hexokinase II to mitochondria by phosphorylating voltage-dependent anion channel and potentiates chemotherapy-induced cytotoxicity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.12.023

10.1016/J.YJMCC.2008.12.023

7 January 2021

Identifiers

DOI

1 reference

31 January 2017

release_vj6pmrpvtnd6jigv4q7uvnxp4a

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/vj6pmrpvtnd6jigv4q7uvnxp4a