(Q34088689)

30 July 2017

The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Abeta protofibril formation (English)

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30 July 2017

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Nilsberth C

1

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30 July 2017

Westlind-Danielsson A

2

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30 July 2017

Eckman CB

3

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30 July 2017

Condron MM

4

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30 July 2017

Axelman K

5

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30 July 2017

Forsell C

6

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30 July 2017

Stenh C

7

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30 July 2017

Luthman J

8

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30 July 2017

Teplow DB

9

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30 July 2017

Younkin SG

10

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30 July 2017

Näslund J

11

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30 July 2017

Lannfelt L

12

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30 July 2017

language of work or name

English

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publication date

1 September 2001

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30 July 2017

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30 July 2017

4

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30 July 2017

887-893

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30 July 2017

9

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https://scigraph.springernature.com/pub.10.1038/nn0901-887

30 July 2017

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cites work

Amyloid, the presenilins and Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Translating cell biology into therapeutic advances in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

A pathogenic mutation for probable Alzheimer's disease in the APP gene at the N-terminus of beta-amyloid

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Excessive production of amyloid beta-protein by peripheral cells of symptomatic and presymptomatic patients carrying the Swedish familial Alzheimer disease mutation

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Increased beta-amyloid release and levels of amyloid precursor protein (APP) in fibroblast cell lines from family members with the Swedish Alzheimer's disease APP670/671 mutation.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Mutation of the Alzheimer's disease amyloid gene in hereditary cerebral hemorrhage, Dutch type.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Presenile dementia and cerebral haemorrhage linked to a mutation at codon 692 of the beta-amyloid precursor protein gene.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Flemish and Dutch mutations in amyloid beta precursor protein have different effects on amyloid beta secretion

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Amyloid beta-protein fibrillogenesis. Detection of a protofibrillar intermediate.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

In vitro studies of amyloid beta-protein fibril assembly and toxicity provide clues to the aetiology of Flemish variant (Ala692-->Gly) Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Novel amyloid precursor protein mutation in an Iowa family with dementia and severe cerebral amyloid angiopathy

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Interaction between A beta(1-42) and A beta(1-40) in Alzheimer's beta-amyloid fibril formation in vitro.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Structural and kinetic features of amyloid beta-protein fibrillogenesis.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Atomic force microscopic imaging of seeded fibril formation and fibril branching by the Alzheimer's disease amyloid-beta protein.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Observation of metastable Abeta amyloid protofibrils by atomic force microscopy

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Amyloid beta-protein fibrillogenesis. Structure and biological activity of protofibrillar intermediates

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Assembly of A beta amyloid protofibrils: an in vitro model for a possible early event in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Protofibrillar intermediates of amyloid beta-protein induce acute electrophysiological changes and progressive neurotoxicity in cortical neurons.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Acceleration of oligomerization, not fibrillization, is a shared property of both alpha-synuclein mutations linked to early-onset Parkinson's disease: implications for pathogenesis and therapy

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

An increased percentage of long amyloid beta protein secreted by familial amyloid beta protein precursor (beta APP717) mutants.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Mutation of the beta-amyloid precursor protein in familial Alzheimer's disease increases beta-protein production

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid beta-protein in both transfected cells and transgenic mice

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Amyloid beta protein in plasma from patients with sporadic Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Amyloid beta protein in plasma as a diagnostic marker for Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Plasma amyloid beta-peptide 1-42 and incipient Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

The cell biology of beta-amyloid precursor protein and presenilin in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

The A beta 3-pyroglutamyl and 11-pyroglutamyl peptides found in senile plaque have greater beta-sheet forming and aggregation propensities in vitro than full-length A beta.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Arrest of beta-amyloid fibril formation by a pentapeptide ligand

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Inhibition of Alzheimer's amyloidosis by peptides that prevent beta-sheet conformation

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Intracellular APP processing and A beta production in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Intraneuronal Abeta42 accumulation in human brain.

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Models of amyloid seeding in Alzheimer's disease and scrapie: mechanistic truths and physiological consequences of the time-dependent solubility of amyloid proteins

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Targeting small Abeta oligomers: the solution to an Alzheimer's disease conundrum?

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Amyloid precursor protein mutation at codon 713 (Ala-->Val) does not cause schizophrenia: non-pathogenic variant found at codon 705 (silent)

1 reference

https://api.crossref.org/works/10.1038%2FNN0901-887

7 January 2021

Identifiers

DOI

10.1038/NN0901-887

1 reference

release_nz3hoz64lbd2rhb74piajdwv4i

30 July 2017

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/nz3hoz64lbd2rhb74piajdwv4i

24 November 2022

mapped directly with Wikidata item

1 reference