(Q35080206)

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Cross-talk between endoplasmic reticulum (ER) stress and the MEK/ERK pathway potentiates apoptosis in human triple negative breast carcinoma cells: role of a dihydropyrimidone, nifetepimine

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Europe PubMed Central

PubMed publication ID

6 August 2017

Cross-talk between endoplasmic reticulum (ER) stress and the MEK/ERK pathway potentiates apoptosis in human triple negative breast carcinoma cells: role of a dihydropyrimidone, nifetepimine (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

endoplasmic reticulum

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breast carcinoma

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based on heuristic

inferred from title

9

object named as

Gopal C Kundu

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Europe PubMed Central

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6 August 2017

author name string

Swatilekha Ghosh

1

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Europe PubMed Central

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6 August 2017

Arghya Adhikary

2

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6 August 2017

Supriya Chakraborty

3

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6 August 2017

Pushpak Bhattacharjee

4

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6 August 2017

Minakshi Mazumder

5

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Europe PubMed Central

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6 August 2017

Salil Putatunda

6

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Europe PubMed Central

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6 August 2017

Mahadeo Gorain

7

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Europe PubMed Central

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6 August 2017

Arijit Chakraborty

8

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Europe PubMed Central

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6 August 2017

Tanya Das

10

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6 August 2017

Parimal C Sen

11

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6 August 2017

language of work or name

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publication date

19 December 2014

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Europe PubMed Central

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6 August 2017

Journal of Biological Chemistry

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Europe PubMed Central

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6 August 2017

290

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Europe PubMed Central

PubMed publication ID

6 August 2017

7

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

3936-3949

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

Nifetepimine, a dihydropyrimidone, ensures CD4+ T cell survival in a tumor microenvironment by maneuvering sarco(endo)plasmic reticulum Ca2+ ATPase (SERCA).

1 reference

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Global cancer statistics

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8 July 2018

Triple-negative breast cancer: an unmet medical need

1 reference

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Treatment of triple negative breast cancer (TNBC): current options and future perspectives

1 reference

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8 July 2018

Triple-negative breast cancer

1 reference

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Triple-negative breast cancer: a short review

1 reference

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Biology, metastatic patterns, and treatment of patients with triple-negative breast cancer

1 reference

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Transcription factor TFII-I causes transcriptional upregulation of GRP78 synthesis in prostate cancer cells

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Triple-negative breast cancer: risk factors to potential targets

1 reference

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Inhibition of MEK sensitizes human melanoma cells to endoplasmic reticulum stress-induced apoptosis

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Triple-negative breast cancer: clinical features and patterns of recurrence

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Targeting the Raf-MEK-ERK mitogen-activated protein kinase cascade for the treatment of cancer

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GRP78 induction in cancer: therapeutic and prognostic implications.

1 reference

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Transcription factor TFII-I conducts a cytoplasmic orchestra

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Roles of the Raf/MEK/ERK pathway in cell growth, malignant transformation and drug resistance

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Expression of stress response protein Grp78 is associated with the development of castration-resistant prostate cancer

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Current strategies in overcoming resistance of cancer cells to apoptosis melanoma as a model.

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8 July 2018

GRP78 as a novel predictor of responsiveness to chemotherapy in breast cancer

1 reference

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Sarco/endoplasmic reticulum Ca2+ATPase type 3 isoforms (SERCA3b and SERCA3f): distinct roles in cell adhesion and ER stress

1 reference

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8 July 2018

Cellular response to endoplasmic reticulum stress: a matter of life or death

1 reference

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8 July 2018

The mammalian unfolded protein response

1 reference

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8 July 2018

Transcriptional regulation of the Grp78 promoter by endoplasmic reticulum stress: role of TFII-I and its tyrosine phosphorylation.

1 reference

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8 July 2018

Molecular mechanisms of drug resistance

1 reference

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8 July 2018

ER stress and the unfolded protein response

1 reference

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The role of the unfolded protein response in tumour development: friend or foe?

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CHOP is involved in endoplasmic reticulum stress-induced apoptosis by enhancing DR5 expression in human carcinoma cells

1 reference

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Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Abeta-induced cell death

1 reference

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Signaling the unfolded protein response from the endoplasmic reticulum.

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Transcriptional and translational control in the Mammalian unfolded protein response

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Organelle-specific initiation of cell death pathways

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Biochemistry and biology of the inducible multifunctional transcription factor TFII-I.

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JAK2 activates TFII-I and regulates its interaction with extracellular signal-regulated kinase.

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Extracellular signal-regulated kinase binds to TFII-I and regulates its activation of the c-fos promoter

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De-regulation of GRP stress protein expression in human breast cancer cell lines.

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The mammalian endoplasmic reticulum stress response element consists of an evolutionarily conserved tripartite structure and interacts with a novel stress-inducible complex

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Regulation of TFII-I activity by phosphorylation

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Mechanisms of apoptosis avoidance in cancer.

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Identifiers

10.1074/JBC.M114.594028

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 August 2017

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