(Q35120714)

6 August 2017

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title

Mitochondrial membrane permeabilisation by Bax/Bak (English)

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6 August 2017

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https://api.crossref.org/works/10.1016/S0006-291X(03)00617-X

30 December 2023

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https://api.crossref.org/works/10.1016/S0006-291X(03)00617-X

30 December 2023

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https://api.crossref.org/works/10.1016/S0006-291X(03)00617-X

30 December 2023

Mauro Degli Esposti

1

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Caroline Dive

object named as

Caroline Dive

2

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language of work or name

English

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publication date

1 May 2003

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6 August 2017

9 May 2003

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National Center for Biotechnology Information

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=12729579

full work available at URL

30 December 2023

https://api.elsevier.com/content/article/PII:S0006291X0300617X?httpAccept=text/xml

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https://api.crossref.org/works/10.1016/S0006-291X(03)00617-X

https://api.elsevier.com/content/article/PII:S0006291X0300617X?httpAccept=text/plain

30 December 2023

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https://api.crossref.org/works/10.1016/S0006-291X(03)00617-X

Biochemical and Biophysical Research Communications

30 December 2023

published in

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6 August 2017

volume

304

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6 August 2017

issue

3

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6 August 2017

page(s)

455-461

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The Bcl2 family: regulators of the cellular life-or-death switch

6 August 2017

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BH-3-only BIK functions at the endoplasmic reticulum to stimulate cytochrome c release from mitochondria

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Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

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7 January 2021

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Damage-induced Bax N-terminal change, translocation to mitochondria and formation of Bax dimers/complexes occur regardless of cell fate

7 January 2021

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

7 January 2021

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

7 January 2021

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Bid, a widely expressed proapoptotic protein of the Bcl-2 family, displays lipid transfer activity.

7 January 2021

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Cytochrome c release upon Fas receptor activation depends on translocation of full-length bid and the induction of the mitochondrial permeability transition

7 January 2021

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Survival factor-mediated BAD phosphorylation raises the mitochondrial threshold for apoptosis

7 January 2021

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Keeping killers on a tight leash: transcriptional and post-translational control of the pro-apoptotic activity of BH3-only proteins

7 January 2021

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Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

7 January 2021

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Breaking the mitochondrial barrier

7 January 2021

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Sequence and functional similarities between pro-apoptotic Bid and plant lipid transfer proteins

7 January 2021

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BCL-2 family: regulators of cell death

7 January 2021

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Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis

7 January 2021

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Cellular damage signals promote sequential changes at the N-terminus and BH-1 domain of the pro-apoptotic protein Bak.

7 January 2021

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BID: a novel BH3 domain-only death agonist

7 January 2021

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

7 January 2021

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Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

7 January 2021

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Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

7 January 2021

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BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

7 January 2021

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BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

7 January 2021

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A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis

7 January 2021

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Spontaneous and drug-induced apoptosis is mediated by conformational changes of Bax and Bak in B-cell chronic lymphocytic leukemia

7 January 2021

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7 January 2021

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7 January 2021

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tBID Homooligomerizes in the mitochondrial membrane to induce apoptosis

7 January 2021

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Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Bax and Bak promote apoptosis by modulating endoplasmic reticular and mitochondrial Ca2+ stores

7 January 2021

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Bax oligomerization in mitochondrial membranes requires tBid (caspase-8-cleaved Bid) and a mitochondrial protein.

7 January 2021

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Regulation of apoptosis by BH3 domains in a cell-free system

7 January 2021

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Dimerization properties of human BAD. Identification of a BH-3 domain and analysis of its binding to mutant BCL-2 and BCL-XL proteins

7 January 2021

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Peptides derived from BH3 domains of Bcl-2 family members: a comparative analysis of inhibition of Bcl-2, Bcl-x(L) and Bax oligomerization, induction of cytochrome c release, and activation of cell death

7 January 2021

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Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

7 January 2021

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Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells

7 January 2021

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Bax, along with lipid conspirators, allows cytochrome c to escape mitochondria

7 January 2021

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Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

7 January 2021

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Tumor necrosis factor-alpha induces Bax-Bak interaction and apoptosis, which is inhibited by adenovirus E1B 19K

7 January 2021

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Bax-type apoptotic proteins porate pure lipid bilayers through a mechanism sensitive to intrinsic monolayer curvature.

7 January 2021

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Direct evidence for membrane pore formation by the apoptotic protein Bax

7 January 2021

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Bax and Bak coalesce into novel mitochondria-associated clusters during apoptosis

7 January 2021

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Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosis

7 January 2021

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7 January 2021

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The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis

7 January 2021

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Loss of OPA1 perturbates the mitochondrial inner membrane structure and integrity, leading to cytochrome c release and apoptosis

7 January 2021

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7 January 2021

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Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

7 January 2021

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The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal Fragment

7 January 2021

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Mitochondrial dynamics and division in budding yeast

7 January 2021

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The expanding role of mitochondria in apoptosis

7 January 2021

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The pro-apoptotic protein, Bik, exhibits potent antitumor activity that is dependent on its BH3 domain

30 December 2023

49

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BH3 death domain peptide induces cell type-selective mitochondrial outer membrane permeability

30 December 2023

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Cell permeable Bcl-2 binding peptides: a chemical approach to apoptosis induction in tumor cells

30 December 2023

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30 December 2023

Identifiers

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6 August 2017

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