(Q37202661)

English

Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8(+) T-cell memory formation and function.

scientific article published on 04 July 2013

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4 March 2020

Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8(+) T-cell memory formation and function (English)

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4 March 2020

memory formation

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based on heuristic

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hyper-IgE syndrome

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based on heuristic

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Capucine Picard

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4 March 2020

Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome

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6 September 2017

Autosomal dominant STAT3 deficiency and hyper-IgE syndrome: molecular, cellular, and clinical features from a French national survey

1 reference

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Inborn errors of human STAT1: allelic heterogeneity governs the diversity of immunological and infectious phenotypes

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6 September 2017

Inborn errors of human JAKs and STATs

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Functional STAT3 deficiency compromises the generation of human T follicular helper cells.

1 reference

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A critical role for STAT3 transcription factor signaling in the development and maintenance of human T cell memory

1 reference

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DOCK8 deficiency impairs CD8 T cell survival and function in humans and mice

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Intrinsic IL-21 signaling is critical for CD8 T cell survival and memory formation in response to vaccinia viral infection

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Cutting edge: The transcription factor eomesodermin enables CD8+ T cells to compete for the memory cell niche.

1 reference

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Diffuse large B cell lymphoma in hyper-IgE syndrome due to STAT3 mutation

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Perforin deficiency and susceptibility to cancer

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CD57+ T lymphocytes and functional immune deficiency.

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IL-21 preferentially enhances IL-15-mediated homeostatic proliferation of human CD28+ CD8 memory T cells throughout the adult age span

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Hyper-IgE syndrome

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Transcriptional repressor Blimp-1 promotes CD8(+) T cell terminal differentiation and represses the acquisition of central memory T cell properties

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Pathogenesis of hyper IgE syndrome

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Homeostasis of naive and memory T cells

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STAT3 mutations in the hyper-IgE syndrome

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Hyper-IgE syndromes

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3785237

3 September 2018

IL-15 Promotes the Survival of Naive and Memory Phenotype CD8+ T Cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3785237

3 September 2018

The role of shared receptor motifs and common Stat proteins in the generation of cytokine pleiotropy and redundancy by IL-2, IL-4, IL-7, IL-13, and IL-15.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3785237

3 September 2018

Bcl6 is essential for the generation of long-term memory CD4+ T cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/23830147

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Impaired survival and proliferation in IL-7 receptor-deficient peripheral T cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/23830147

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

IL-21 promotes differentiation of naive CD8 T cells to a unique effector phenotype

1 reference

https://pubmed.ncbi.nlm.nih.gov/23830147

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

IL-21 promotes T lymphocyte survival by activating the phosphatidylinositol-3 kinase signaling cascade

1 reference

https://pubmed.ncbi.nlm.nih.gov/23830147

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

IL-21 limits NK cell responses and promotes antigen-specific T cell activation: a mediator of the transition from innate to adaptive immunity

1 reference

https://pubmed.ncbi.nlm.nih.gov/23830147

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1016/J.JACI.2013.05.029

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23830147%20AND%20SRC:MED&resulttype=core&format=json

4 March 2020

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23830147%20AND%20SRC:MED&resulttype=core&format=json

4 March 2020

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23830147%20AND%20SRC:MED&resulttype=core&format=json

4 March 2020

ResearchGate publication ID

0 references

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