(Q37646370)

21 August 2017

1 reference

title

The PINK1/Parkin pathway: a mitochondrial quality control system? (English)

1 reference

21 August 2017

0 references

1 reference

Alexander J. Whitworth

object named as

Alexander J Whitworth

series ordinal

1

0 references

author name string

Leo J Pallanck

series ordinal

2

1 reference

21 August 2017

1 December 2009

1 reference

Journal of Bioenergetics and Biomembranes

21 August 2017

1 reference

21 August 2017

41

1 reference

21 August 2017

499-503

1 reference

21 August 2017

6

1 reference

https://scigraph.springernature.com/pub.10.1007/s10863-009-9253-3

21 August 2017

0 references

cites work

Parkinson's disease mutations in PINK1 result in decreased Complex I activity and deficient synaptic function

1 reference

12 December 2020

The PINK1/Parkin pathway regulates mitochondrial morphology

1 reference

12 December 2020

Mechanistic approaches to Parkinson's disease pathogenesis.

1 reference

12 December 2020

Loss-of-function of human PINK1 results in mitochondrial pathology and can be rescued by parkin

1 reference

12 December 2020

Cytoplasmic Pink1 activity protects neurons from dopaminergic neurotoxin MPTP.

1 reference

12 December 2020

Impaired dopamine release and synaptic plasticity in the striatum of PINK1-deficient mice

1 reference

12 December 2020

Ubiquitin signals autophagic degradation of cytosolic proteins and peroxisomes

1 reference

12 December 2020

Parkinson's disease: from causes to mechanisms

1 reference

12 December 2020

Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants

1 reference

12 December 2020

Mitochondrial dysfunction in Parkinson's disease

1 reference

12 December 2020

Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability

1 reference

12 December 2020

Rhomboid-7 and HtrA2/Omi act in a common pathway with the Parkinson's disease factors Pink1 and Parkin

1 reference

12 December 2020

PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1

1 reference

12 December 2020

Synaptic mitochondria are critical for mobilization of reserve pool vesicles at Drosophila neuromuscular junctions

1 reference

12 December 2020

Absence of nigral degeneration in aged parkin/DJ-1/PINK1 triple knockout mice

1 reference

12 December 2020

Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons.

1 reference

12 December 2020

PINK1-associated Parkinson's disease is caused by neuronal vulnerability to calcium-induced cell death

1 reference

12 December 2020

DJ-1 and Parkin modulate dopamine-dependent behavior and inhibit MPTP-induced nigral dopamine neuron loss in mice

1 reference

12 December 2020

Molecular pathogenesis of Parkinson disease: insights from genetic studies

1 reference

12 December 2020

High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease

1 reference

12 December 2020

Inclusion body formation and neurodegeneration are parkin independent in a mouse model of alpha-synucleinopathy.

1 reference

12 December 2020

The kinase domain of mitochondrial PINK1 faces the cytoplasm

1 reference

12 December 2020

Parkin-deficient mice exhibit nigrostriatal deficits but not loss of dopaminergic neurons

1 reference

12 December 2020

Loss of parkin or PINK1 function increases Drp1-dependent mitochondrial fragmentation

1 reference

12 December 2020

Silencing of PINK1 expression affects mitochondrial DNA and oxidative phosphorylation in dopaminergic cells

1 reference

12 December 2020

Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

1 reference

12 December 2020

Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin

1 reference

12 December 2020

Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

1 reference

12 December 2020

Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin

1 reference

12 December 2020

The Parkinson's disease genes pink1 and parkin promote mitochondrial fission and/or inhibit fusion in Drosophila

1 reference

12 December 2020

Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin

1 reference

12 December 2020

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

1 reference

12 December 2020

Impaired dopamine release and synaptic plasticity in the striatum of parkin-/- mice.

1 reference

12 December 2020

Mitochondrial alterations in PINK1 deficient cells are influenced by calcineurin-dependent dephosphorylation of dynamin-related protein 1.

1 reference

12 December 2020

Functions and dysfunctions of mitochondrial dynamics

1 reference

12 December 2020

Expanding insights of mitochondrial dysfunction in Parkinson's disease.

1 reference

12 December 2020

Pink1 Parkinson mutations, the Cdc37/Hsp90 chaperones and Parkin all influence the maturation or subcellular distribution of Pink1.

1 reference

12 December 2020

Parkin protects against mitochondrial toxins and beta-amyloid accumulation in skeletal muscle cells.

1 reference

12 December 2020

Pink1 regulates mitochondrial dynamics through interaction with the fission/fusion machinery

1 reference

12 December 2020

PINK1 protein in normal human brain and Parkinson's disease

1 reference

12 December 2020

The PINK1-Parkin pathway is involved in the regulation of mitochondrial remodeling process.

1 reference

12 December 2020

Characterization of PINK1 processing, stability, and subcellular localization

1 reference

12 December 2020

10.1007/S10863-009-9253-3

Identifiers

DOI

1 reference

21 August 2017

1 reference

PubMed ID

19967438