(Q38244247)

31 August 2017

Natural polyphenols binding to amyloid: a broad class of compounds to treat different human amyloid diseases (English)

1 reference

31 August 2017

1 reference

Viviane L Ndam Ngoungoure

1

1 reference

31 August 2017

Jan Schluesener

2

1 reference

31 August 2017

Paul F Moundipa

3

1 reference

31 August 2017

Hermann Schluesener

4

1 reference

31 August 2017

9 October 2014

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Molecular Nutrition and Food Research

31 August 2017

1 reference

31 August 2017

59

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31 August 2017

1

1 reference

31 August 2017

8-20

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Chemistry and biochemistry of dietary polyphenols

31 August 2017

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Dietary phenolics: chemistry, bioavailability and effects on health

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Herbal extracts and phytochemicals: plant secondary metabolites and the enhancement of human brain function

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Fruit and vegetable intake and risk of major chronic disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Dietary polyphenols as potential nutraceuticals in management of diabetes: a review

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Immunopathogenesis of neurodegenerative diseases: current therapeutic models of neuroprotection with special reference to natural products

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Protein folding and aggregation into amyloid: the interference by natural phenolic compounds

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Molecular and cellular aspects of protein misfolding and disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Molecular basis for amyloid fibril formation and stability

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Inhibition of amyloid fibril formation by polyphenols: structural similarity and aromatic interactions as a common inhibition mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Amyloid-beta fibrillogenesis: structural insight and therapeutic intervention

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The workings of the amyloid diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Review: history of the amyloid fibril

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Mechanisms of amyloid fibril self-assembly and inhibition. Model short peptides as a key research tool

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Amyloid fibrils: abnormal protein assembly

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Amyloid beta-protein assembly and Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Soluble protein oligomers as emerging toxins in Alzheimer's and other amyloid diseases.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Structural classification of toxic amyloid oligomers

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

A new human hereditary amyloidosis: the result of a stop-codon mutation in the apolipoprotein AII gene

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Light-chain (AL) amyloidosis: diagnosis and treatment

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Gelsolin amyloidosis: genetics, biochemistry, pathology and possible strategies for therapeutic intervention

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Amyloidosis: review and CT manifestations.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Beta2-microglobulin and amyloidosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

α-Synuclein in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The function of α-synuclein

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

An overview of human prion diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Clinical review 102: Type 2 diabetes mellitus: update on diagnosis, pathophysiology, and treatment

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Islet amyloid and type 2 diabetes: from molecular misfolding to islet pathophysiology

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Type 2 diabetes and the aging pancreatic beta cell

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Islet amyloid polypeptide, islet amyloid, and diabetes mellitus

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Islet amyloid in type 2 diabetes, and the toxic oligomer hypothesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Natural biomolecules and protein aggregation: emerging strategies against amyloidogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Inhibiting toxic aggregation of amyloidogenic proteins: a therapeutic strategy for protein misfolding diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Polyphenols as potential inhibitors of amyloid aggregation and toxicity: possible significance to Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Natural polyphenols as inhibitors of amyloid aggregation. Molecular dynamics study of GNNQQNY heptapeptide decamer.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Natural polyphenols against neurodegenerative disorders: potentials and pitfalls

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Polyphenols: multipotent therapeutic agents in neurodegenerative diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Phenol red interacts with the protofibril-like oligomers of an amyloidogenic hexapeptide NFGAIL through both hydrophobic and aromatic contacts.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Comparative study of inhibition at multiple stages of amyloid-beta self-assembly provides mechanistic insight.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Inhibition and disaggregation of α-synuclein oligomers by natural polyphenolic compounds

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Inhibition of islet amyloid polypeptide fibril formation: a potential role for heteroaromatic interactions

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Phenolic compounds prevent amyloid β-protein oligomerization and synaptic dysfunction by site-specific binding

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin decreases amyloid-beta peptide levels by attenuating the maturation of amyloid-beta precursor protein.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin inhibits aggregation of alpha-synuclein

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The effect of curcumin on human islet amyloid polypeptide misfolding and toxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin has potent anti-amyloidogenic effects for Alzheimer's beta-amyloid fibrils in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Effect of curcumin on the amyloid fibrillogenesis of hen egg-white lysozyme.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin inhibits formation of amyloid beta oligomers and fibrils, binds plaques, and reduces amyloid in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin modulates α-synuclein aggregation and toxicity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin prevents aggregation in α-synuclein by increasing reconfiguration rate.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Natural polyphenols inhibit different steps of the process of transthyretin (TTR) amyloid fibril formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Resveratrol inhibits beta-amyloid oligomeric cytotoxicity but does not prevent oligomer formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Resveratrol inhibits the formation of multiple-layered β-sheet oligomers of the human islet amyloid polypeptide segment 22-27

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Comparision of piceid and resveratrol in antioxidation and antiproliferation activities in vitro.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The polyphenol piceid destabilizes preformed amyloid fibrils and oligomers in vitro: hypothesis on possible molecular mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The spice sage and its active ingredient rosmarinic acid protect PC12 cells from amyloid-beta peptide-induced neurotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Phenolic compounds prevent Alzheimer's pathology through different effects on the amyloid-beta aggregation pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Ferulic acid destabilizes preformed beta-amyloid fibrils in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Baicalein reduces β-amyloid and promotes nonamyloidogenic amyloid precursor protein processing in an Alzheimer's disease transgenic mouse model

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The flavonoid baicalein inhibits fibrillation of alpha-synuclein and disaggregates existing fibrils

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Structural characteristics of alpha-synuclein oligomers stabilized by the flavonoid baicalein

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Anti-amyloidogenic activity of tannic acid and its activity to destabilize Alzheimer's beta-amyloid fibrils in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Antioxidant compounds have potent anti-fibrillogenic and fibril-destabilizing effects for alpha-synuclein fibrils in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Green tea epigallocatechin-3-gallate (EGCG) modulates amyloid precursor protein cleavage and reduces cerebral amyloidosis in Alzheimer transgenic mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

EGCG remodels mature alpha-synuclein and amyloid-beta fibrils and reduces cellular toxicity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Green tea (-)-epigallocatechin-gallate modulates early events in huntingtin misfolding and reduces toxicity in Huntington's disease models

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

The flavanol (-)-epigallocatechin 3-gallate inhibits amyloid formation by islet amyloid polypeptide, disaggregates amyloid fibrils, and protects cultured cells against IAPP-induced toxicity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Potent anti-amyloidogenic and fibril-destabilizing effects of polyphenols in vitro: implications for the prevention and therapeutics of Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Myricetin Inhibits Islet Amyloid Polypeptide (IAPP) Aggregation and Rescues Living Mammalian Cells from IAPP Toxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Morin hydrate inhibits amyloid formation by islet amyloid polypeptide and disaggregates amyloid fibers

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Salvianolic acid B inhibits Abeta fibril formation and disaggregates preformed fibrils and protects against Abeta-induced cytotoxicty.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Salvianolic acid B inhibits the amyloid formation of human islet amyloid polypeptide and protects pancreatic beta-cells against cytotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Oleuropein aglycon prevents cytotoxic amyloid aggregation of human amylin.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Silibinin attenuates amyloid beta(25-35) peptide-induced memory impairments: implication of inducible nitric-oxide synthase and tumor necrosis factor-alpha in mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Silibinin prevents amyloid beta peptide-induced memory impairment and oxidative stress in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Silibinin: a novel inhibitor of Aβ aggregation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Silibinin inhibits the toxic aggregation of human islet amyloid polypeptide

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Ellagic acid promotes Abeta42 fibrillization and inhibits Abeta42-induced neurotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

New stilbene dimers against amyloid fibril formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

New polyphenols active on beta-amyloid aggregation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Curcumin structure-function, bioavailability, and efficacy in models of neuroinflammation and Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

A chemical analog of curcumin as an improved inhibitor of amyloid Abeta oligomerization

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Amyloid-binding compounds maintain protein homeostasis during ageing and extend lifespan

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Role of protein misfolding and proteostasis deficiency in protein misfolding diseases and aging

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

SPECT Imaging Agents for Detecting Cerebral β-Amyloid Plaques

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Amyloid tracers detect multiple binding sites in Alzheimer's disease brain tissue.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Aβ Imaging: feasible, pertinent, and vital to progress in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Novel radioiodinated aurones as probes for SPECT imaging of beta-amyloid plaques in the brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Novel chalcones as probes for in vivo imaging of beta-amyloid plaques in Alzheimer's brains.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Aurones serve as probes of beta-amyloid plaques in Alzheimer's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Radioiodinated flavones for in vivo imaging of beta-amyloid plaques in the brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Novel stilbenes as probes for amyloid plaques.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Polyphenols as therapeutic molecules in Alzheimer's disease through modulating amyloid pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Dietary polyphenol-derived protection against neurotoxic β-amyloid protein: from molecular to clinical.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Ferulic acid is a nutraceutical β-secretase modulator that improves behavioral impairment and alzheimer-like pathology in transgenic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

Oridonin ameliorates neuropathological changes and behavioural deficits in a mouse model of cerebral amyloidosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FMNFR.201400290

21 January 2018

Identifiers

DOI

10.1002/MNFR.201400290

1 reference

31 August 2017

1 reference