(Q38258607)

1 September 2017

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title

The role of autophagic degradation in the heart (English)

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1 September 2017

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Kazuhiko Nishida

1

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1 September 2017

Manabu Taneike

series ordinal

2

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1 September 2017

Kinya Otsu

series ordinal

3

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1 September 2017

language of work or name

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6 October 2014

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Journal of Molecular and Cellular Cardiology

1 September 2017

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1 September 2017

78

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1 September 2017

73-79

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The autophagosome: origins unknown, biogenesis complex

1 September 2017

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

The role of Atg proteins in autophagosome formation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagy: renovation of cells and tissues

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

The role of autophagy during the early neonatal starvation period

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitochondrial homeostasis: the interplay between mitophagy and mitochondrial biogenesis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

7 January 2021

Mechanisms of mitophagy

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

During autophagy mitochondria elongate, are spared from degradation and sustain cell viability.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagosomes form at ER-mitochondria contact sites

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagosome formation from membrane compartments enriched in phosphatidylinositol 3-phosphate and dynamically connected to the endoplasmic reticulum

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Atg9 vesicles are an important membrane source during early steps of autophagosome formation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

At the end of the autophagic road: an emerging understanding of lysosomal functions in autophagy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

A gene network regulating lysosomal biogenesis and function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

The hairpin-type tail-anchored SNARE syntaxin 17 targets to autophagosomes for fusion with endosomes/lysosomes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Atg8-family interacting motif crucial for selective autophagy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitochondria-anchored receptor Atg32 mediates degradation of mitochondria via selective autophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Atg32 is a mitochondrial protein that confers selectivity during mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Role of BNIP3 and NIX in cell death, autophagy, and mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Nix is a selective autophagy receptor for mitochondrial clearance

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Microtubule-associated protein 1 light chain 3 (LC3) interacts with Bnip3 protein to selectively remove endoplasmic reticulum and mitochondria via autophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitochondrial outer-membrane protein FUNDC1 mediates hypoxia-induced mitophagy in mammalian cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Hereditary early-onset Parkinson's disease caused by mutations in PINK1

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitofusin 1 and mitofusin 2 are ubiquitinated in a PINK1/parkin-dependent manner upon induction of mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitochondria and mitophagy: the yin and yang of cell death control

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Disease-causing mutations in parkin impair mitochondrial ubiquitination, aggregation, and HDAC6-dependent mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

p62/SQSTM1 is required for Parkin-induced mitochondrial clustering but not mitophagy; VDAC1 is dispensable for both

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

A role for NBR1 in autophagosomal degradation of ubiquitinated substrates

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Bcl-2 family proteins participate in mitochondrial quality control by regulating Parkin/PINK1-dependent mitophagy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Parkin interacts with Ambra1 to induce mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Differential expression and tissue distribution of parkin isoforms during mouse development

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Novel monoclonal antibodies demonstrate biochemical variation of brain parkin with age.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Ultrastructural morphometric analysis of myocardium from dogs, rats, hamsters, mice, and from human hearts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Discovery of Atg5/Atg7-independent alternative macroautophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Ulk1-mediated Atg5-independent macroautophagy mediates elimination of mitochondria from embryonic reticulocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Alterations in mitochondrial function in cardiac hypertrophy and heart failure.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Histone deacetylase (HDAC) inhibitors attenuate cardiac hypertrophy by suppressing autophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagy-mediated degradation is necessary for regression of cardiac hypertrophy during ventricular unloading

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Reversal of cardiac hypertrophy by medical treatment

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Left ventricular mass regression early after aortic valve replacement

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Regression of cellular hypertrophy after left ventricular assist device support

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Reversal of left ventricular hypertrophy in essential hypertension. A meta-analysis of randomized double-blind studies

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagy plays an essential role in mediating regression of hypertrophy during unloading of the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mechanical unloading activates FoxO3 to trigger Bnip3-dependent cardiomyocyte atrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Metabolic remodelling of the failing heart: the cardiac burn-out syndrome?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

The role of autophagy in the heart.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitochondrial DNA that escapes from autophagy causes inflammation and heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Cardiac autophagy is a maladaptive response to hemodynamic stress

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagy in load-induced heart disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Lysosomal glycogen storage disease with normal acid maltase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Danon disease: focusing on heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Accumulation of autophagic vacuoles and cardiomyopathy in LAMP-2-deficient mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Primary LAMP-2 deficiency causes X-linked vacuolar cardiomyopathy and myopathy (Danon disease).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Autophagy is an adaptive response in desmin-related cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Atg7 induces basal autophagy and rescues autophagic deficiency in CryABR120G cardiomyocytes.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Enhanced autophagy ameliorates cardiac proteinopathy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Diminished autophagy limits cardiac injury in mouse models of type 1 diabetes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Contribution of impaired mitochondrial autophagy to cardiac aging: mechanisms and therapeutic opportunities

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Inhibition of autophagy in the heart induces age-related cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Overexpression of Atg5 in mice activates autophagy and extends lifespan

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Mitochondrial quality control in the myocardium: cooperation between protein degradation and mitophagy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Cytosolic p53 inhibits Parkin-mediated mitophagy and promotes mitochondrial dysfunction in the mouse heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

Parkin protein deficiency exacerbates cardiac injury and reduces survival following myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.09.029

10.1016/J.YJMCC.2014.09.029

7 January 2021

Identifiers

DOI

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1 September 2017

1 reference