(Q40503168)

21 September 2017

title

The apoptotic pathway triggered by the Fhit protein in lung cancer cell lines is not affected by Bcl-2 or Bcl-x(L) overexpression (English)

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21 September 2017

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4

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21 September 2017

Francesca Andriani

2

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Luca Roz

object named as

Luca Roz

series ordinal

1

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Gabriella Sozzi

object named as

Gabriella Sozzi

series ordinal

5

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author name string

Carlos G Ferreira

series ordinal

3

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21 September 2017

language of work or name

English

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publication date

1 December 2004

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21 September 2017

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21 September 2017

volume

23

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21 September 2017

issue

56

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21 September 2017

page(s)

9102-9110

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1208142

21 September 2017

exact match

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cites work

Loss of FHIT expression in transitional cell carcinoma of the urinary bladder

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Signal transduction by DR3, a death domain-containing receptor related to TNFR-1 and CD95

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Inhibition of apoptosis and clonogenic survival of cells expressing crmA variants: optimal caspase substrates are not necessarily optimal inhibitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

The biochemistry of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Bcl-2, Bcl-XL and adenovirus protein E1B19kD are functionally equivalent in their ability to inhibit cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Potential cancer therapy with the fragile histidine triad gene: review of the preclinical studies

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Apoptosis: a link between cancer genetics and chemotherapy

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Programmed cell death: alive and well in the new millennium

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Mortality from smoking worldwide.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Restoration of fragile histidine triad (FHIT) expression induces apoptosis and suppresses tumorigenicity in lung and cervical cancer cell lines

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

The tumor-suppressor gene FHIT is involved in the regulation of apoptosis and in cell cycle control

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

Differential modulation of apoptosis sensitivity in CD95 type I and type II cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208142

21 January 2018

Identifiers

DOI

10.1038/SJ.ONC.1208142

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Dimensions Publication ID

21 September 2017

1009726858

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1 reference