(Q42952019)

English

Reversible cyclosporin A-sensitive mitochondrial depolarization occurs within minutes of stroke onset in mouse somatosensory cortex in vivo: a two-photon imaging study

scientific article published on 5 November 2009

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

Reversible cyclosporin A-sensitive mitochondrial depolarization occurs within minutes of stroke onset in mouse somatosensory cortex in vivo: a two-photon imaging study (English)

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

author name string

Ran R Liu

1

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

Timothy H Murphy

2

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

language of work or name

0 references

publication date

5 November 2009

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

Journal of Biological Chemistry

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Europe PubMed Central

PMC publication ID

11 November 2017

284

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

52

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

36109-36117

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC2794726/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Intracellular Zn2+ accumulation contributes to synaptic failure, mitochondrial depolarization, and cell death in an acute slice oxygen-glucose deprivation model of ischemia.

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Pathophysiology and therapy of experimental stroke

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Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

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In vivo two-photon calcium imaging of neuronal networks

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Cyclosporin A-insensitive permeability transition in brain mitochondria: inhibition by 2-aminoethoxydiphenyl borate

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Cyclosporin A, but not FK 506, protects mitochondria and neurons against hypoglycemic damage and implicates the mitochondrial permeability transition in cell death.

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Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase

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Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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Identifiers

10.1074/JBC.M109.055301

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

11 November 2017

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