(Q44143951)

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Detection of elevated levels of soluble alpha-synuclein oligomers in post-mortem brain extracts from patients with dementia with Lewy bodies.

scientific article published on 20 January 2009

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20 January 2020

Detection of elevated levels of soluble alpha-synuclein oligomers in post-mortem brain extracts from patients with dementia with Lewy bodies (English)

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20 January 2020

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20 January 2020

Poul Henning Jensen

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20 January 2020

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Katerina E Paleologou

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20 January 2020

Christine L Kragh

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20 January 2020

David M A Mann

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20 January 2020

Sultan A Salem

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20 January 2020

Rania Al-Shami

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20 January 2020

Ahmed H Hassan

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20 January 2020

Omar M A El-Agnaf

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20 January 2020

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20 January 2009

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20 January 2020

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20 January 2020

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20 January 2020

Pt 4

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20 January 2020

1093-1101

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20 January 2020

Inhibitors of alpha-synuclein oligomerization and toxicity: a future therapeutic strategy for Parkinson's disease and related disorders

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Neuropathological stageing of Alzheimer-related changes

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Alpha-synuclein locus duplication as a cause of familial Parkinson's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

The biochemistry of Parkinson's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Effects of the mutations Ala30 to Pro and Ala53 to Thr on the physical and morphological properties of alpha-synuclein protein implicated in Parkinson's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Review: formation and properties of amyloid-like fibrils derived from alpha-synuclein and related proteins

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Aggregation and neurotoxicity of alpha-synuclein and related peptides

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Oligomerization and toxicity of beta-amyloid-42 implicated in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Non-fibrillar oligomeric species of the amyloid ABri peptide, implicated in familial British dementia, are more potent at inducing apoptotic cell death than protofibrils or mature fibrils

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

A strategy for designing inhibitors of alpha-synuclein aggregation and toxicity as a novel treatment for Parkinson's disease and related disorders

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Alpha-synuclein implicated in Parkinson's disease is present in extracellular biological fluids, including human plasma.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Soluble oligomers for the diagnosis of neurodegenerative diseases

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Binding of Abeta to alpha- and beta-synucleins: identification of segments in alpha-synuclein/NAC precursor that bind Abeta and NAC

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Microtubule-associated protein 1B is a component of cortical Lewy bodies and binds alpha-synuclein filaments

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Selective insolubility of alpha-synuclein in human Lewy body diseases is recapitulated in a transgenic mouse model

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Clinical and biochemical correlates of insoluble alpha-synuclein in dementia with Lewy bodies.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Amyloid beta protein immunotherapy neutralizes Abeta oligomers that disrupt synaptic plasticity in vivo.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Ala30Pro mutation in the gene encoding alpha-synuclein in Parkinson's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Alpha-synuclein, especially the Parkinson's disease-associated mutants, forms pore-like annular and tubular protofibrils

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Proteasomal inhibition by alpha-synuclein filaments and oligomers.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

p25alpha Stimulates alpha-synuclein aggregation and is co-localized with aggregated alpha-synuclein in alpha-synucleinopathies

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

alpha -Synucleinopathy and selective dopaminergic neuron loss in a rat lentiviral-based model of Parkinson's disease.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Dopaminergic loss and inclusion body formation in alpha-synuclein mice: implications for neurodegenerative disorders

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Consensus guidelines for the clinical and pathologic diagnosis of dementia with Lewy bodies (DLB): report of the consortium on DLB international workshop

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part II. Standardization of the neuropathologic assessment of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Alpha-synuclein aggregation in neurodegenerative diseases and its inhibition as a potential therapeutic strategy

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Senile dementia of Lewy body type. A clinically and neuropathologically distinct form of Lewy body dementia in the elderly

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Mutation in the alpha-synuclein gene identified in families with Parkinson's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Cell biology of protein misfolding: the examples of Alzheimer's and Parkinson's diseases

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

alpha-Synuclein locus triplication causes Parkinson's disease

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodies

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Alpha-synuclein in Lewy bodies

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Hydrogen peroxide is generated during the very early stages of aggregation of the amyloid peptides implicated in Alzheimer disease and familial British dementia.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Certain inhibitors of synthetic amyloid beta-peptide (Abeta) fibrillogenesis block oligomerization of natural Abeta and thereby rescue long-term potentiation.

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

The new mutation, E46K, of alpha-synuclein causes Parkinson and Lewy body dementia

1 reference

https://api.crossref.org/works/10.1093%2FBRAIN%2FAWN349

21 January 2018

Identifiers

10.1093/BRAIN/AWN349

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19155272%20AND%20SRC:MED&resulttype=core&format=json

20 January 2020

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19155272%20AND%20SRC:MED&resulttype=core&format=json

20 January 2020

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