(Q44279382)

1 December 2017

Possible dominant-negative mutation of the SHIP gene in acute myeloid leukemia (English)

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1 December 2017

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J-M Luo

1

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1 December 2017

H Yoshida

2

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1 December 2017

S Komura

3

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1 December 2017

N Ohishi

4

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1 December 2017

L Pan

5

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1 December 2017

K Shigeno

6

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1 December 2017

I Hanamura

7

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1 December 2017

K Miura

8

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1 December 2017

S Iida

9

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1 December 2017

R Ueda

10

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1 December 2017

T Naoe

11

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1 December 2017

Y Akao

12

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1 December 2017

R Ohno

13

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1 December 2017

K Ohnishi

14

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1 December 2017

1 January 2003

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1 December 2017

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1 December 2017

17

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1 December 2017

1-8

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1 December 2017

1

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https://scigraph.springernature.com/pub.10.1038/sj.leu.2402725

1 December 2017

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cites work

Oncogenic kinase signalling

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Protein kinase B (c-Akt): a multifunctional mediator of phosphatidylinositol 3-kinase activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Transcription-dependent and -independent control of neuronal survival by the PI3K-Akt signaling pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Induction of apoptosis by ASK1, a mammalian MAPKKK that activates SAPK/JNK and p38 signaling pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

The tumor suppressor, PTEN/MMAC1, dephosphorylates the lipid second messenger, phosphatidylinositol 3,4,5-trisphosphate

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Mutational analysis of the tumour suppressor gene MMAC1/PTEN in malignant myeloid disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

SHIP is a negative regulator of growth factor receptor-mediated PKB/Akt activation and myeloid cell survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

BCR/ABL directly inhibits expression of SHIP, an SH2-containing polyinositol-5-phosphatase involved in the regulation of hematopoiesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

'Cold SSCP': a simple, rapid and non-radioactive method for optimized single-strand conformation polymorphism analyses

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

The 145-kDa protein induced to associate with Shc by multiple cytokines is an inositol tetraphosphate and phosphatidylinositol 3,4,5-triphosphate 5-phosphatase

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Properties of type II inositol polyphosphate 5-phosphatase

21 January 2018

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https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Interleukin-3 induces the association of the inositol 5-phosphatase SHIP with SHP2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Targeted disruption of SHIP leads to hemopoietic perturbations, lung pathology, and a shortened life span

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

The src homology 2-containing inositol phosphatase (SHIP) is the gatekeeper of mast cell degranulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Specific activation of p85-p110 phosphatidylinositol 3'-kinase stimulates DNA synthesis by ras- and p70 S6 kinase-dependent pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Microinjection of the SH2 domain of the 85-kilodalton subunit of phosphatidylinositol 3-kinase inhibits insulin-induced DNA synthesis and c-fos expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

PTEN/MMAC1/TEP1 suppresses the tumorigenicity and induces G1 cell cycle arrest in human glioblastoma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Alternative signals to RAS for hematopoietic transformation by the BCR-ABL oncogene

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

PTEN expression is reduced in a subset of sporadic thyroid carcinomas: evidence that PTEN-growth suppressing activity in thyroid cancer cells mediated by p27kip1

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Forkhead transcription factors are critical effectors of cell death and cell cycle arrest downstream of PTEN.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

The Src homology 2 (SH2) domain of SH2-containing inositol phosphatase (SHIP) is essential for tyrosine phosphorylation of SHIP, its association with Shc, and its induction of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

BCL-2 is phosphorylated and inactivated by an ASK1/Jun N-terminal protein kinase pathway normally activated at G(2)/M.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Akt phosphorylates and negatively regulates apoptosis signal-regulating kinase 1

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

The lipid phosphatase activity of PTEN is critical for its tumor supressor function

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

Dual role of phosphatidylinositol-3,4,5-trisphosphate in the activation of protein kinase B

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

High cancer susceptibility and embryonic lethality associated with mutation of the PTEN tumor suppressor gene in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.LEU.2402725

21 January 2018

Identifiers

DOI

10.1038/SJ.LEU.2402725

1 reference

1 December 2017

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