(Q44384541)

English

No alterations of hippocampal neuronal number and synaptic bouton number in a transgenic mouse model expressing the beta-cleaved C-terminal APP fragment

scientific article published in March 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

2 December 2017

No alterations of hippocampal neuronal number and synaptic bouton number in a transgenic mouse model expressing the beta-cleaved C-terminal APP fragment (English)

1 reference

Europe PubMed Central

PubMed publication ID

2 December 2017

Harry W M Steinbusch

6

object named as

Harry W M Steinbusch

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Europe PubMed Central

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2 December 2017

1

object named as

Bart P F Rutten

0 references

Konrad Beyreuther

8

object named as

Konrad Beyreuther

1 reference

Europe PubMed Central

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2 December 2017

9

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Gerd Multhaup

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Europe PubMed Central

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2 December 2017

Stefan F Lichtenthaler

4

object named as

Stefan F Lichtenthaler

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2 December 2017

author name string

Bart P F Rutten

1

1 reference

Europe PubMed Central

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2 December 2017

Oliver Wirths

2

1 reference

Europe PubMed Central

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2 December 2017

Wilma D J Van de Berg

3

1 reference

Europe PubMed Central

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2 December 2017

Jochen Vehoff

5

1 reference

Europe PubMed Central

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2 December 2017

Hubert Korr

7

1 reference

Europe PubMed Central

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2 December 2017

Thomas A Bayer

10

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2 December 2017

Christoph Schmitz

11

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2 December 2017

publication date

1 March 2003

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2 December 2017

Neurobiology of Disease

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2 December 2017

12

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Europe PubMed Central

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2 December 2017

2

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Europe PubMed Central

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2 December 2017

110-120

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Europe PubMed Central

PubMed publication ID

2 December 2017

C-terminal fragment of amyloid precursor protein induces astrocytosis

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Impairments in learning and memory accompanied by neurodegeneration in mice transgenic for the carboxyl-terminus of the amyloid precursor protein.

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Neuronal deficiency of presenilin 1 inhibits amyloid plaque formation and corrects hippocampal long-term potentiation but not a cognitive defect of amyloid precursor protein [V717I] transgenic mice.

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Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

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Membrane inserted APP fragments containing the beta A4 sequence of Alzheimer's disease do not aggregate

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Prion protein (PrP) with amino-proximal deletions restoring susceptibility of PrP knockout mice to scrapie.

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Selective neurotoxicity of COOH-terminal fragments of the beta-amyloid precursor protein

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Presenilins as therapeutic targets for the treatment of Alzheimer's disease

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Intraneuronal Abeta42 accumulation in human brain.

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The efficiency of systematic sampling in stereology and its prediction.

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Analysis of heterogeneous A4 peptides in human cerebrospinal fluid and blood by a newly developed sensitive Western blot assay.

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The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor

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Intracellular amyloid-beta 1-42, but not extracellular soluble amyloid-beta peptides, induces neuronal apoptosis

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Neuron loss during early adulthood following prenatal low-dose X-irradiation in the mouse brain

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The neurotoxic carboxy-terminal fragment of the Alzheimer amyloid precursor binds specifically to a neuronal cell surface molecule: pH dependence of the neurotoxicity and the binding

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Candidate gene for the chromosome 1 familial Alzheimer's disease locus

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A novel substrate for analyzing Alzheimer's disease gamma-secretase

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Mechanism of the cleavage specificity of Alzheimer's disease gamma-secretase identified by phenylalanine-scanning mutagenesis of the transmembrane domain of the amyloid precursor protein

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Amyloid plaque core protein in Alzheimer disease and Down syndrome

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7 January 2021

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PC12 cells transfected with a C-terminal fragment of the amyloid precursor protein (APP C-100), exhibit enhanced sensitivity to the calcium ionophore A23187, and diminished sensitivity to hydrogen peroxide

1 reference

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7 January 2021

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beta-Secretase cleavage of the amyloid precursor protein mediates neuronal apoptosis caused by familial Alzheimer's disease mutations

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A pathogenic mutation for probable Alzheimer's disease in the APP gene at the N-terminus of beta-amyloid

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7 January 2021

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Age-dependent neuronal and synaptic degeneration in mice transgenic for the C terminus of the amyloid precursor protein.

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Familial Alzheimer's disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer's disease type 3 gene

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Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

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1 reference

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Intraneuronal APP/A beta trafficking and plaque formation in beta-amyloid precursor protein and presenilin-1 transgenic mice.

1 reference

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Identifiers

10.1016/S0969-9961(02)00015-3

1 reference

Europe PubMed Central

PubMed publication ID

2 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 December 2017

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