(Q46740612)

24 December 2017

title

Targeting toxic proteins for turnover (English)

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24 December 2017

2

Patrick Weydt

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24 December 2017

author name string

Albert R La Spada

series ordinal

1

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24 December 2017

language of work or name

English

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publication date

1 October 2005

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24 December 2017

published in

Nature Medicine

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24 December 2017

volume

11

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24 December 2017

issue

10

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24 December 2017

page(s)

1052-1053

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24 December 2017

https://scigraph.springernature.com/pub.10.1038/nm1005-1052

exact match

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cites work

Toxic proteins in neurodegenerative disease

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

17-AAG, an Hsp90 inhibitor, ameliorates polyglutamine-mediated motor neuron degeneration

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Cellular defenses against unfolded proteins: a cell biologist thinks about neurodegenerative diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Polyglutamines placed into context

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Eukaryotic proteasomes cannot digest polyglutamine sequences and release them during degradation of polyglutamine-containing proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Inefficient degradation of truncated polyglutamine proteins by the proteasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Effect of geldanamycin on androgen receptor function and stability

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Pharmacologic shifting of a balance between protein refolding and degradation mediated by Hsp90

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Recovery from polyglutamine-induced neurodegeneration in conditional SCA1 transgenic mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

Reversal of neuropathology and motor dysfunction in a conditional model of Huntington's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNM1005-1052

21 January 2018

Identifiers

DOI

10.1038/NM1005-1052

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24 December 2017

1 reference

24 December 2017